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Protein kinase N1 deficiency results in upregulation of cerebral energy metabolism and is highly protective in in vivo and in vitro stroke models
by
zur Nedden, Stephanie
,
Safari, Motahareh S.
,
Weber, Dido
,
Kuenkel, Louisa
,
Garmsiri, Carolin
,
Lang, Luisa
,
Orset, Cyrille
,
Freret, Tom
,
Haelewyn, Benoît
,
Hotze, Madlen
,
Kwiatkowski, Marcel
,
Sarg, Bettina
,
Faserl, Klaus
,
Savic, Dragana
,
Skvortsova, Ira-Ida
,
Krogsdam, Anne
,
Carollo, Sandro
,
Trajanoski, Zlatko
,
Oberacher, Herbert
,
Zlotek, Dominik
,
Ostermaier, Florian
,
Cameron, Angus
,
Baier, Gottfried
,
Baier-Bitterlich, Gabriele
Published in
Metabolism, clinical and experimental
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Metabolism, Clinical And Experimental
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Ampk
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Animals
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Brain - Metabolism
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Cerebral Energy Metabolism
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Disease Models, Animal
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Endocrinology & Metabolism
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Energy Metabolism
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Glucose - Metabolism
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Life Sciences & Biomedicine
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Male
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Mice
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Mice, Inbred C57Bl
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Mice, Knockout
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Mitochondria - Metabolism
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Oxygen Consumption
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Pfk
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Pgm2L1
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Protein Kinase C - Genetics
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Protein Kinase C - Metabolism
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Protein Kinase N1
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