Search Results - Geenen, Sarah R

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  1. 1
    Fluorescent Donor–Acceptor Psoralen Cruciforms by Consecutive Suzuki–Suzuki and Sonogashira–Sonogashira One-Pot Syntheses

    Fluorescent Donor–Acceptor Psoralen Cruciforms by Consecutive Suzuki–Suzuki and Sonogashira–Sonogashira One-Pot Syntheses by Geenen, Sarah R, Schumann, Torben, Müller, Thomas J. J

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  2. 2
    Electronic Finetuning of 8‐Methoxy Psoralens by Palladium‐Catalyzed Coupling: Acidochromicity and Solvatochromicity

    Electronic Finetuning of 8‐Methoxy Psoralens by Palladium‐Catalyzed Coupling: Acidochromicity and Solvatochromicity by Geenen, Sarah R., Presser, Lysander, Hölzel, Torsten, Ganter, Christian, Müller, Thomas J. J.

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  3. 3
    Ablation of Ventricular Myosin Regulatory Light Chain Phosphorylation in Mice Causes Cardiac Dysfunction in Situ and Affects Neighboring Myofilament Protein Phosphorylation

    Ablation of Ventricular Myosin Regulatory Light Chain Phosphorylation in Mice Causes Cardiac Dysfunction in Situ and Affects Neighboring Myofilament Protein Phosphorylation by Scruggs, Sarah B., Hinken, Aaron C., Thawornkaiwong, Ariyaporn, Robbins, Jeffrey, Walker, Lori A., de Tombe, Pieter P., Geenen, David L., Buttrick, Peter M., Solaro, R. John

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  4. 4
    Myocardial infarction in mice alters sarcomeric function via post-translational protein modification

    Myocardial infarction in mice alters sarcomeric function via post-translational protein modification by Avner, Benjamin S., Shioura, Krystyna M., Scruggs, Sarah B., Grachoff, Milana, Geenen, David L., Helseth, Donald L., Farjah, Mariam, Goldspink, Paul H., John Solaro, R.

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  5. 5
    Tuning and external validation of an adult congenital heart disease risk prediction model

    Tuning and external validation of an adult congenital heart disease risk prediction model by Geenen, Laurie W, Opotowsky, Alexander R, Lachtrupp, Cara, Baggen, Vivan J M, Brainard, Sarah, Landzberg, Michael J, van Klaveren, David, Lingsma, Hester F, Boersma, Eric, Roos-Hesselink, Jolien W

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  6. 6
    Partial replacement of cardiac troponin I with a non-phosphorylatable mutant at serines 43/45 attenuates the contractile dysfunction associated with PKCε phosphorylation

    Partial replacement of cardiac troponin I with a non-phosphorylatable mutant at serines 43/45 attenuates the contractile dysfunction associated with PKCε phosphorylation by Scruggs, Sarah B., Walker, Lori A., Lyu, Theodore, Geenen, David L., Solaro, R. John, Buttrick, Peter M., Goldspink, Paul H.

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  7. 7
    Partial replacement of cardiac troponin I with a non-phosphorylatable mutant at serines 43/45 attenuates the contractile dysfunction associated with PKCepsilon phosphorylation

    Partial replacement of cardiac troponin I with a non-phosphorylatable mutant at serines 43/45 attenuates the contractile dysfunction associated with PKCepsilon phosphorylation by Scruggs, Sarah B, Walker, Lori A, Lyu, Theodore, Geenen, David L, Solaro, R John, Buttrick, Peter M, Goldspink, Paul H

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