Search Results - HAUSEL, Pierrette

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  1. 1
    Nedd4-2 Modulates Renal Na+-Cl― Cotransporter via the Aldosterone-SGK1-Nedd4-2 Pathway

    Nedd4-2 Modulates Renal Na+-Cl― Cotransporter via the Aldosterone-SGK1-Nedd4-2 Pathway by PABLO ARROYO, Juan, LAGNAZ, Dagmara, STOKES, John B, HOOVER, Robert S, GAMBA, Gerardo, STAUB, Olivier, RONZAUD, Caroline, VAZQUEZ, Norma, KO, Benjamin S, MODDES, Lauren, RUFFIEUX-DAIDIE, Dorothée, HAUSEL, Pierrette, KOESTERS, Robert, YANG, Baoli

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  2. 2
    Renal tubular NEDD4-2 deficiency causes NCC-mediated salt-dependent hypertension

    Renal tubular NEDD4-2 deficiency causes NCC-mediated salt-dependent hypertension by Ronzaud, Caroline, Loffing-Cueni, Dominique, Hausel, Pierrette, Debonneville, Anne, Malsure, Sumedha Ram, Fowler-Jaeger, Nicole, Boase, Natasha A, Perrier, Romain, Maillard, Marc, Yang, Baoli, Stokes, John B, Koesters, Robert, Kumar, Sharad, Hummler, Edith, Loffing, Johannes, Staub, Olivier

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  3. 3
    Nedd4-2 modulates renal Na+-Cl- cotransporter via the aldosterone-SGK1-Nedd4-2 pathway

    Nedd4-2 modulates renal Na+-Cl- cotransporter via the aldosterone-SGK1-Nedd4-2 pathway by Arroyo, Juan Pablo, Lagnaz, Dagmara, Ronzaud, Caroline, Vázquez, Norma, Ko, Benjamin S, Moddes, Lauren, Ruffieux-Daidié, Dorothée, Hausel, Pierrette, Koesters, Robert, Yang, Baoli, Stokes, John B, Hoover, Robert S, Gamba, Gerardo, Staub, Olivier

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  4. 4
    Transition mutation in codon 248 of the p53 tumor suppressor gene induced by reactive oxygen species and a nitric oxide-releasing compound

    Transition mutation in codon 248 of the p53 tumor suppressor gene induced by reactive oxygen species and a nitric oxide-releasing compound by Souici, Anne-Christine, Mirkovitch, Jovan, Hausel, Pierrette, K.Keefer, Larry, Felley-Bosco, Emanuela

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  5. 5
    Studies on possible mechanisms of early functional compensatory adaptation in the remaining kidney

    Studies on possible mechanisms of early functional compensatory adaptation in the remaining kidney by Diezi, J, Michoud-Hausel, P, Nicolas-Buxcel, N

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