Search Results - Hu, Rou‐Mu

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  1. 1
    Study on the relationship between telomere length changes and recurrence of atrial fibrillation after radiofrequency catheter ablation

    Study on the relationship between telomere length changes and recurrence of atrial fibrillation after radiofrequency catheter ablation by Su, Changyang, Liu, Zheng, Gao, Yuanfeng, Liu, Ye, Hu, Rou‐Mu, Liu, Jia, Yang, Xiyan, Li, Shichao, Zhang, Yuan, Zuo, Kun, Cao, Boli, Luo, Jing, Li, Jing, Li, Kuibao, Yin, Xiandong, Chen, Mulei, Yang, Xinchun

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  2. 2
    Arrhythmogenic Biophysical Phenotype for SCN5A Mutation S1787N Depends upon Splice Variant Background and Intracellular Acidosis

    Arrhythmogenic Biophysical Phenotype for SCN5A Mutation S1787N Depends upon Splice Variant Background and Intracellular Acidosis by Hu, Rou-Mu, Tan, Bi-Hua, Tester, David J, Song, Chunhua, He, Yang, Dovat, Sinisa, Peterson, Blaise Z, Ackerman, Michael J, Makielski, Jonathan C

    Published in PloS one
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  3. 3
    Investigation of the Cellular Pharmacological Mechanism and Clinical Evidence of the Multi-Herbal Antiarrhythmic Chinese Medicine Xin Su Ning

    Investigation of the Cellular Pharmacological Mechanism and Clinical Evidence of the Multi-Herbal Antiarrhythmic Chinese Medicine Xin Su Ning by Ma, Yu-Ling, Hu, Rou-Mu, Yang, Xinchun, Wang, Taiyi, Noble, Penelope J, Wilkins, Robert, Ellory, Clive, Carr, Carolyn, Noble, Denis, Yang, Jiefu, Lu, Weixing, Zhang, Junhua, Hu, Hongde, Guo, Xiaomei, Chen, Min, Wu, Yang, Wei, Meng, Mao, Jingyuan, Ma, Xiaochang, Qin, Ling, Wu, Huanlin, Lu, Feng, Cao, Ying, Gao, Sheng, Gu, Wanli

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  4. 4
    Mexiletine rescues a mixed biophysical phenotype of the cardiac sodium channel arising from the SCN5A mutation, N406K, found in LQT3 patients

    Mexiletine rescues a mixed biophysical phenotype of the cardiac sodium channel arising from the SCN5A mutation, N406K, found in LQT3 patients by Hu, Rou-Mu, Tester, David J., Li, Ryan, Sun, Tianyu, Peterson, Blaise Z., Ackerman, Michael J., Makielski, Jonathan C., Tan, Bi-Hua

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  5. 5
    Expression defect of the rare variant/Brugada mutation R1512W depends upon the SCN5A splice variant background and can be rescued by mexiletine and the common polymorphism H558R

    Expression defect of the rare variant/Brugada mutation R1512W depends upon the SCN5A splice variant background and can be rescued by mexiletine and the common polymorphism H558R by Hu, Rou-Mu, Song, Evelyn J., Tester, David J., Deschenes, Isabelle, Ackerman, Michael J., Makielski, Jonathan C., Tan, Bi-Hua

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  6. 6
    Digenic inheritance novel mutations in SCN5a and SNTA1 increase late INa contributing to LQT syndrome

    Digenic inheritance novel mutations in SCN5a and SNTA1 increase late INa contributing to LQT syndrome by Hu, Rou-Mu, Tan, Bi-Hua, Orland, Kate M., Valdivia, Carmen R., Peterson, Amber, Pu, Jielin, Makielski, Jonathan C.

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  7. 7
    Trafficking-Defective Kir6.1 (KATP) Mutations in Sudden Infant Death Syndrome

    Trafficking-Defective Kir6.1 (KATP) Mutations in Sudden Infant Death Syndrome by Tan, Bi-Hua, Hu, Rou-Mu, Peterson, Blaise, Dovat, Sinisa, Ackerman, Michael J., Makielski, Jonathan C., Song, Chunhua

    Published in Biophysical journal
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  8. 8
    Digenic inheritance novel mutations in SCN5a and SNTA1 increase late I Na contributing to LQT syndrome

    Digenic inheritance novel mutations in SCN5a and SNTA1 increase late I Na contributing to LQT syndrome by Hu, Rou-Mu, Tan, Bi-Hua, Orland, Kate M., Valdivia, Carmen R., Peterson, Amber, Pu, Jielin, Makielski, Jonathan C.

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  9. 9
    Digenic inheritance novel mutations in SCN5a and SNTA1 increase late /^sub Na^ contributing to LQT syndrome

    Digenic inheritance novel mutations in SCN5a and SNTA1 increase late /^sub Na^ contributing to LQT syndrome by Hu, Rou-Mu, Tan, Bi-Hua, Orland, Kate M, Valdivia, Carmen R, Peterson, Amber, Pu, Jielin, Makielski, Jonathan C

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  10. 10
    Arrhythmogenic Biophysical Phenotype for SCN5A Mutation S1787N Depends upon Splice Variant Background and Intracellular Acidosis: e0124921

    Arrhythmogenic Biophysical Phenotype for SCN5A Mutation S1787N Depends upon Splice Variant Background and Intracellular Acidosis: e0124921 by Hu, Rou-Mu, Tan, Bi-Hua, Tester, David J, Song, Chunhua, He, Yang, Dovat, Sinisa, Peterson, Blaise Z, Ackerman, Michael J, Makielski, Jonathan C

    Published in PloS one
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  11. 11
    Digenic inheritance novel mutations in SCN5a and SNTA1 increase late I(Na) contributing to LQT syndrome

    Digenic inheritance novel mutations in SCN5a and SNTA1 increase late I(Na) contributing to LQT syndrome by Hu, Rou-Mu, Tan, Bi-Hua, Orland, Kate M, Valdivia, Carmen R, Peterson, Amber, Pu, Jielin, Makielski, Jonathan C

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