Cytogenetic complexity in chronic lymphocytic leukemia: definitions, associations, and clinical impact by Baliakas, Panagiotis, Jeromin, Sabine, Iskas, Michalis, Puiggros, Anna, Plevova, Karla, Nguyen-Khac, Florence, Davis, Zadie, Rigolin, Gian Matteo, Visentin, Andrea, Xochelli, Aliki, Delgado, Julio, Baran-Marszak, Fanny, Stalika, Evangelia, Abrisqueta, Pau, Durechova, Kristina, Papaioannou, George, Eclache, Virginie, Dimou, Maria, Iliakis, Theodoros, Collado, Rosa, Doubek, Michael, Calasanz, M. Jose, Ruiz-Xiville, Neus, Moreno, Carolina, Jarosova, Marie, Leeksma, Alexander C., Panayiotidis, Panayiotis, Podgornik, Helena, Cymbalista, Florence, Anagnostopoulos, Achilles, Trentin, Livio, Stavroyianni, Niki, Davi, Fred, Ghia, Paolo, Kater, Arnon P., Cuneo, Antonio, Pospisilova, Sarka, Espinet, Blanca, Athanasiadou, Anastasia, Oscier, David, Haferlach, Claudia, Stamatopoulos, Kostas

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The mutational landscape of 18 investigated genes clearly separates four subtypes of myelodysplastic/myeloproliferative neoplasms by Meggendorfer, Manja, Jeromin, Sabine, Haferlach, Claudia, Kern, Wolfgang, Haferlach, Torsten

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Indeterminate and oncogenic potential: CHIP vs CHOP mutations in AML with NPM1 alteration by Cappelli, Luca Vincenzo, Meggendorfer, Manja, Baer, Constance, Nadarajah, Niroshan, Hutter, Stephan, Jeromin, Sabine, Dicker, Frank, Kern, Wolfgang, Haferlach, Torsten, Haferlach, Claudia, Höllein, Alexander

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Specific molecular mutation patterns delineate chronic neutrophilic leukemia, atypical chronic myeloid leukemia, and chronic myelomonocytic leukemia by Meggendorfer, Manja, Haferlach, Torsten, Alpermann, Tamara, Jeromin, Sabine, Haferlach, Claudia, Kern, Wolfgang, Schnittger, Susanne

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Detection and characterization of homozygosity of mutated CALR by copy neutral loss of heterozygosity in myeloproliferative neoplasms among cases with high CALR mutation loads or w... by Stengel, Anna, Jeromin, Sabine, Haferlach, Torsten, Meggendorfer, Manja, Kern, Wolfgang, Haferlach, Claudia

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Refractory anemia with ring sideroblasts and marked thrombocytosis cases harbor mutations in SF3B1 or other spliceosome genes accompanied by JAK2V617F and ASXL1 mutations by Jeromin, Sabine, Haferlach, Torsten, Weissmann, Sandra, Meggendorfer, Manja, Eder, Christiane, Nadarajah, Niroshan, Alpermann, Tamara, Kohlmann, Alexander, Kern, Wolfgang, Haferlach, Claudia, Schnittger, Susanne

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Tailored approaches grounded on immunogenetic features for refined prognostication in chronic lymphocytic leukemia by Baliakas, Panagiotis, Moysiadis, Theodoros, Hadzidimitriou, Anastasia, Xochelli, Aliki, Jeromin, Sabine, Agathangelidis, Andreas, Mattsson, Mattias, Sutton, Lesley-Ann, Minga, Eva, Scarfò, Lydia, Rossi, Davide, Davis, Zadie, Villamor, Neus, Parker, Helen, Kotaskova, Jana, Stalika, Evangelia, Plevova, Karla, Mansouri, Larry, Cortese, Diego, Navarro, Alba, Delgado, Julio, Larrayoz, Marta, Young, Emma, Anagnostopoulos, Achilles, Smedby, Karin E, Juliusson, Gunnar, Sheehy, Oonagh, Catherwood, Mark, Strefford, Jonathan C, Stavroyianni, Niki, Belessi, Chrysoula, Pospisilova, Sarka, Oscier, David, Gaidano, Gianluca, Campo, Elias, Haferlach, Claudia, Ghia, Paolo, Rosenquist, Richard, Stamatopoulos, Kostas

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High frequencies of SF3B1 and JAK2 mutations in refractory anemia with ring sideroblasts associated with marked thrombocytosis strengthen the assignment to the category of myelodys... by Jeromin, Sabine, Haferlach, Torsten, Grossmann, Vera, Alpermann, Tamara, Kowarsch, Andreas, Haferlach, Claudia, Kern, Wolfgang, Schnittger, Susanne

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Acute myeloid leukemias with ring sideroblasts show a unique molecular signature straddling secondary acute myeloid leukemia and de novo acute myeloid leukemia by Martin-Cabrera, Pedro, Jeromin, Sabine, Perglerovà, Karolína, Haferlach, Claudia, Kern, Wolfgang, Haferlach, Torsten

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Prognostic relevance of RUNX1 mutations in T-cell acute lymphoblastic leukemia by GROSSMANN, Vera, KERN, Wolfgang, HARBICH, Stefan, ALPERMANN, Tamara, JEROMIN, Sabine, SCHNITTGER, Susanne, HAFERLACH, Claudia, HAFERLACH, Torsten, KOHLMANN, Alexander

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T cell receptor gene repertoire profiles in subgroups of patients with chronic lymphocytic leukemia bearing distinct genomic aberrations by Vlachonikola, Elisavet, Pechlivanis, Nikolaos, Karakatsoulis, Georgios, Sofou, Electra, Gkoliou, Glykeria, Jeromin, Sabine, Stavroyianni, Niki, Ranghetti, Pamela, Scarfo, Lydia, Österholm, Cecilia, Mansouri, Larry, Notopoulou, Sofia, Siorenta, Alexandra, Anagnostopoulos, Achilles, Ghia, Paolo, Haferlach, Claudia, Rosenquist, Richard, Psomopoulos, Fotis, Kouvatsi, Anastasia, Baliakas, Panagiotis, Stamatopoulos, Kostas, Chatzidimitriou, Anastasia

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In Ph+BCR-ABL1P210+ acute lymphoblastic leukemia the e13a2 (B2A2) transcript is prevalent by Baccarani, Michele, Iacobucci, Ilaria, Chiaretti, Sabina, Foà’, Robin, Balasubramanian, Poonkuzhali, Paietta, Elisabeth, Foroni, Letizia, Jeromin, Sabine, Izzo, Barbara, Spinelli, Orietta, Varma, Neelam, Menif, Samia, Terragna, Carolina, Seth, Tulika, Bidet, Audrey, Coriu, Daniel, Lunghi, Francesca, Mayer, Jiri, Scappini, Barbara, Langabeer, Stephen, Maier, Jacqueline, Burt, Emma, Candoni, Anna, Albano, Francesco, Luppi, Mario, Zupan, Irena, Lion, Thomas, Zadro, Renata, di Raimondo, Francesco, Poopak, Behzad, Rege-Cambrin, Giovanna, Annunziata, Mario, Ayala, Ana, Salinas-Viedma, Victor, Ines Prado, Ana, Milner, Benedict, Galimberti, Sara, Janssen, Jeroen, Polli, Valentina, Comba, Lorenzo, Borsellino, Beatrice, Annibali, Ombretta, Crugnola, Monica, Passamonti, Francesco

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Molecular characterization of AML with RUNX1‐RUNX1T1 at diagnosis and relapse reveals net loss of co‐mutations by Höllein, Alexander, Nadarajah, Niroshan, Meggendorfer, Manja, Jeromin, Sabine, Kern, Wolfgang, Haferlach, Claudia, Haferlach, Torsten

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Higher-order connections between stereotyped subsets: implications for improved patient classification in CLL by Agathangelidis, Andreas, Chatzidimitriou, Anastasia, Gemenetzi, Katerina, Giudicelli, Veronique, Karypidou, Maria, Plevova, Karla, Davis, Zadie, Yan, Xiao-Jie, Jeromin, Sabine, Schneider, Christof, Pedersen, Lone Bredo, Tschumper, Renee C., Sutton, Lesley-Ann, Baliakas, Panagiotis, Scarfò, Lydia, van Gastel, Ellen J., Armand, Marine, Tausch, Eugen, Biderman, Bella, Baer, Constance, Bagnara, Davide, Navarro, Alba, Langlois de Septenville, Anne, Guido, Valentina, Mitterbauer-Hohendanner, Gerlinde, Dimovski, Aleksandar, Brieghel, Christian, Lawless, Sarah, Meggendorfer, Manja, Brazdilova, Kamila, Ritgen, Matthias, Facco, Monica, Tresoldi, Cristina, Visentin, Andrea, Patriarca, Andrea, Catherwood, Mark, Bonello, Lisa, Sudarikov, Andrey, Vanura, Katrina, Roumelioti, Maria, Skuhrova Francova, Hana, Moysiadis, Theodoros, Veronese, Silvio, Giannopoulos, Krzysztof, Mansouri, Larry, Karan-Djurasevic, Teodora, Sandaltzopoulos, Raphael, Bödör, Csaba, Fais, Franco, Kater, Arnon, Panovska, Irina, Rossi, Davide, Alshemmari, Salem, Panagiotidis, Panagiotis, Costeas, Paul, Espinet, Blanca, Antic, Darko, Foroni, Letizia, Montillo, Marco, Trentin, Livio, Stavroyianni, Niki, Gaidano, Gianluca, Francia di Celle, Paola, Niemann, Carsten, Campo, Elias, Anagnostopoulos, Achilles, Pott, Christiane, Fischer, Kirsten, Hallek, Michael, Oscier, David, Stilgenbauer, Stephan, Haferlach, Claudia, Jelinek, Diane, Chiorazzi, Nicholas, Pospisilova, Sarka, Lefranc, Marie-Paule, Kossida, Sofia, Langerak, Anton W., Belessi, Chrysoula, Davi, Frederic, Rosenquist, Richard, Ghia, Paolo, Stamatopoulos, Kostas

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Impact of Infrequently Mutated Genes on Prognosis in 531 Untreated CLL Patients by Jeromin, Sabine, Meggendorfer, Manja, Haferlach, Claudia, Haferlach, Torsten, Kern, Wolfgang

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Clonal Evolution of Molecular and Cytogenetic Aberrations in 80 CLL Patients by Jeromin, Sabine, Meggendorfer, Manja, Haferlach, Claudia, Haferlach, Torsten, Kern, Wolfgang

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Minimal residual disease (MRD) monitoring and mutational landscape in AML with RUNX1-RUNX1T1: a study on 134 patients by Höllein, Alexander, Jeromin, Sabine, Meggendorfer, Manja, Fasan, Annette, Nadarajah, Niroshan, Kern, Wolfgang, Haferlach, Claudia, Haferlach, Torsten

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Clonal evolution in chronic lymphocytic leukemia is associated with an unmutated IGHV status and frequently leads to a combination of loss of TP53 and TP53 mutation by Bracher, Susanne, Fuhrmann, Irene, Jeromin, Sabine, Nadarajah, Niroshan, Kern, Wolfgang, Haferlach, Torsten, Haferlach, Claudia, Stengel, Anna

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DNMT3A mutations are over-represented in young adults with NPM1 mutated AML and prompt a distinct co-mutational pattern by Cappelli, Luca Vincenzo, Meggendorfer, Manja, Dicker, Frank, Jeromin, Sabine, Hutter, Stephan, Kern, Wolfgang, Haferlach, Torsten, Haferlach, Claudia, Höllein, Alexander

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Impact of assay procedures on detection of MR4.5 status in chronic myeloid leukemia: Optimization of cDNA synthesis by Jeromin, Sabine, Eder, Christiane, Haferlach, Claudia, Haferlach, Torsten, Kern, Wolfgang

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