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Type 2 Diabetes and Congenital Hyperinsulinism Cause DNA Double-Strand Breaks and p53 Activity in β Cells
by
Tornovsky-Babeay, Sharona
,
Dadon, Daniela
,
Ziv, Oren
,
Tzipilevich, Elhanan
,
Kadosh, Tehila
,
Schyr-Ben Haroush, Rachel
,
Hija, Ayat
,
Stolovich-Rain, Miri
,
Furth-Lavi, Judith
,
Granot, Zvi
,
Porat, Shay
,
Philipson, Louis H.
,
Herold, Kevan C.
,
Bhatti, Tricia R.
,
Stanley, Charles
,
Ashcroft, Frances M.
,
In’t Veld, Peter
,
Saada, Ann
,
Magnuson, Mark A.
,
Glaser, Benjamin
,
Dor, Yuval
Published in
Cell metabolism
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Cell Metabolism
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Animals
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Biomarkers - Metabolism
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Calcineurin - Metabolism
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Cell Biology
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Cell Death - Drug Effects
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Cell Proliferation - Drug Effects
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Congenital Hyperinsulinism - Complications
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Congenital Hyperinsulinism - Enzymology
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Congenital Hyperinsulinism - Pathology
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Diabetes Mellitus, Type 2 - Complications
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Diabetes Mellitus, Type 2 - Enzymology
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Diabetes Mellitus, Type 2 - Pathology
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Disease Models, Animal
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Dna Breaks, Double-Stranded - Drug Effects
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Endocrinology & Metabolism
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Enzyme Activation - Drug Effects
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Enzyme Induction - Drug Effects
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Fasting - Metabolism
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Glucagon-Like Peptide 1 - Pharmacology
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Glucokinase - Biosynthesis
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Bacon - Elsevier - Global Sciencedirect-Openaccess
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