Bioaccumulation and toxicity of gold nanoparticles after repeated administration in mice by Lasagna-Reeves, C., Gonzalez-Romero, D., Barria, M.A., Olmedo, I., Clos, A., Sadagopa Ramanujam, V.M., Urayama, A., Vergara, L., Kogan, M.J., Soto, C.

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Targeting tau protein stability in alzheimer disease to identify novel therapeutic entry points by Lasagna-Reeves, C, Al Ramahi, I, de Haro, M, Rousseaux, M, Kim, J, Jafar-Nejad, P, Vilanova-Velez, L, Westbrook, T, Botas, J, Zoghbi, H

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A native interactor scaffolds and stabilizes toxic ATAXIN-1 oligomers in SCA1 by Lasagna-Reeves, Cristian A, Rousseaux, Maxime Wc, Guerrero-Muñoz, Marcos J, Park, Jeehye, Jafar-Nejad, Paymaan, Richman, Ronald, Lu, Nan, Sengupta, Urmi, Litvinchuk, Alexandra, Orr, Harry T, Kayed, Rakez, Zoghbi, Huda Y

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Poloxamer 188 Copolymer Membrane Sealant Rescues Toxicity of Amyloid Oligomers In Vitro by Mina, Erene W., Lasagna-Reeves, Cristian, Glabe, Charles G., Kayed, Rakez

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Reduction of Nuak1 Decreases Tau and Reverses Phenotypes in a Tauopathy Mouse Model by Lasagna-Reeves, Cristian A., de Haro, Maria, Hao, Shuang, Park, Jeehye, Rousseaux, Maxime W.C., Al-Ramahi, Ismael, Jafar-Nejad, Paymaan, Vilanova-Velez, Luis, See, Lauren, De Maio, Antonia, Nitschke, Larissa, Wu, Zhenyu, Troncoso, Juan C., Westbrook, Thomas F., Tang, Jianrong, Botas, Juan, Zoghbi, Huda Y.

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TREM2 splice isoforms generate soluble TREM2 species that disrupt long-term potentiation by Moutinho, Miguel, Coronel, Israel, Tsai, Andy P, Di Prisco, Gonzalo Viana, Pennington, Taylor, Atwood, Brady K, Puntambekar, Shweta S, Smith, Daniel C, Martinez, Pablo, Han, Seonggyun, Lee, Younghee, Lasagna-Reeves, Cristian A, Lamb, Bruce T, Bissel, Stephanie J, Nho, Kwangsik, Landreth, Gary E

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Loss of Inpp5d has disease‐relevant and sex‐specific effects on glial transcriptomes by Dabin, Luke C., Kersey, Holly, Kim, Byungwook, Acri, Dominic J., Sharify, Daniel, Lee‐Gosselin, Audrey, Lasagna‐Reeves, Cristian A., Oblak, Adrian L., Lamb, Bruce T., Kim, Jungsu

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TRIM28 regulates the nuclear accumulation and toxicity of both alpha-synuclein and tau by Rousseaux, Maxime Wc, de Haro, Maria, Lasagna-Reeves, Cristian A, De Maio, Antonia, Park, Jeehye, Jafar-Nejad, Paymaan, Al-Ramahi, Ismael, Sharma, Ajay, See, Lauren, Lu, Nan, Vilanova-Velez, Luis, Klisch, Tiemo J, Westbrook, Thomas F, Troncoso, Juan C, Botas, Juan, Zoghbi, Huda Y

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Enhanced microglial dynamics and a paucity of tau seeding in the amyloid plaque microenvironment contribute to cognitive resilience in Alzheimer’s disease by Jury-Garfe, Nur, Redding-Ochoa, Javier, You, Yanwen, Martínez, Pablo, Karahan, Hande, Chimal-Juárez, Enrique, Johnson, Travis S., Zhang, Jie, Resnick, Susan, Kim, Jungsu, Troncoso, Juan C., Lasagna-Reeves, Cristian A.

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The formation of tau pore-like structures is prevalent and cell specific: possible implications for the disease phenotypes by Lasagna-Reeves, Cristian A, Sengupta, Urmi, Castillo-Carranza, Diana, Gerson, Julia E, Guerrero-Munoz, Marcos, Troncoso, Juan C, Jackson, George R, Kayed, Rakez

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Ataxin-1 oligomers induce local spread of pathology and decreasing them by passive immunization slows Spinocerebellar ataxia type 1 phenotypes by Lasagna-Reeves, Cristian A, Rousseaux, Maxime Wc, Guerrero-Munoz, Marcos J, Vilanova-Velez, Luis, Park, Jeehye, See, Lauren, Jafar-Nejad, Paymaan, Richman, Ronald, Orr, Harry T, Kayed, Rakez, Zoghbi, Huda Y

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