Search Results - Ngian, Zhen Kai

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  1. 1
    Increased intron retention is a post‐transcriptional signature associated with progressive aging and Alzheimer’s disease

    Increased intron retention is a post‐transcriptional signature associated with progressive aging and Alzheimer’s disease by Adusumalli, Swarnaseetha, Ngian, Zhen‐Kai, Lin, Wei‐Qi, Benoukraf, Touati, Ong, Chin‐Tong

    Published in Aging cell
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  2. 2
    NELF‐A controls Drosophila healthspan by regulating heat‐shock protein‐mediated cellular protection and heterochromatin maintenance

    NELF‐A controls Drosophila healthspan by regulating heat‐shock protein‐mediated cellular protection and heterochromatin maintenance by Ngian, Zhen‐Kai, Lin, Wei‐Qi, Ong, Chin‐Tong

    Published in Aging cell
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  3. 3
    Regulation of aging by NELF-A and RNA polymerase II elongation

    Regulation of aging by NELF-A and RNA polymerase II elongation by Ong, Chin-Tong, Ngian, Zhen-Kai

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  4. 4
    ApoE maintains neuronal integrity via microRNA and H3K27me3-mediated repression

    ApoE maintains neuronal integrity via microRNA and H3K27me3-mediated repression by Tan, Jiazi, Tan, Yow-Yong, Ngian, Zhen-Kai, Chong, Suet-Yen, Rao, Vinay Kumar, Wang, Jiong-Wei, Zeng, Xianmin, Ong, Chin-Tong

    Published in iScience
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  5. 5
    Altered stability of nuclear lamin-B marks the onset of aging in male Drosophila

    Altered stability of nuclear lamin-B marks the onset of aging in male Drosophila by Lin, Wei-Qi, Ngian, Zhen-Kai, Koh, Tong-Wey, Ong, Chin-Tong

    Published in PloS one
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  6. 6
    A neurodegeneration checkpoint mediated by REST protects against the onset of Alzheimer’s disease

    A neurodegeneration checkpoint mediated by REST protects against the onset of Alzheimer’s disease by Aron, Liviu, Qiu, Chenxi, Ngian, Zhen Kai, Liang, Marianna, Drake, Derek, Choi, Jaejoon, Fernandez, Marty A., Roche, Perle, Bunting, Emma L., Lacey, Ella K., Hamplova, Sara E., Yuan, Monlan, Wolfe, Michael S., Bennett, David A., Lee, Eunjung A., Yankner, Bruce A.

    Published in Nature communications
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  7. 7
    Truncated Tau caused by intron retention is enriched in Alzheimer's disease cortex and exhibits altered biochemical properties

    Truncated Tau caused by intron retention is enriched in Alzheimer's disease cortex and exhibits altered biochemical properties by Ngian, Zhen-Kai, Tan, Yow-Yong, Choo, Ching-Thong, Lin, Wei-Qi, Leow, Chao-Yong, Mah, Shan-Jie, Lai, Mitchell Kim-Peng, Chen, Christopher Li-Hsian, Ong, Chin-Tong

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  8. 8
    E2F and STAT3 provide transcriptional synergy for histone variant H2AZ activation to sustain glioblastoma chromatin accessibility and tumorigenicity

    E2F and STAT3 provide transcriptional synergy for histone variant H2AZ activation to sustain glioblastoma chromatin accessibility and tumorigenicity by Yoon, Jeehyun, Grinchuk, Oleg V., Tirado-Magallanes, Roberto, Ngian, Zhen Kai, Tay, Emmy Xue Yun, Chuah, You Heng, Lee, Bernice Woon Li, Feng, Jia, Crasta, Karen Carmelina, Ong, Chin Tong, Benoukraf, Touati, Ong, Derrick Sek Tong

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