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E1A overcomes the apoptosis block in BCR-ABL+ leukemia cells and renders cells susceptible to induction of apoptosis by chemotherapeutic agents
by
STIEWE, T
,
PARSSANEDJAD, K
,
ESCHE, H
,
OPALKA, B
,
PÜTZER, B. M
Published in
Cancer research (Chicago, Ill.)
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Cancer Research
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Adenovirus E1A Proteins - Metabolism
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Antibiotics, Antineoplastic - Pharmacology
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Antineoplastic Agents
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Apoptosis - Drug Effects
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Biological And Medical Sciences
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Blotting, Western
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Caspase 3
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Caspases - Metabolism
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Cdc2 Protein Kinase - Metabolism
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Cell Cycle - Drug Effects
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Cell Division
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Chemotherapy
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Daunorubicin - Pharmacology
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Dna Damage - Drug Effects
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Dose-Response Relationship, Drug
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Etoposide - Pharmacology
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Flow Cytometry
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Fusion Proteins, Bcr-Abl - Metabolism
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Humans
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K562 Cells
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