Search Results - Riemslagh, F W

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  1. 1
    Intact neuronal function in Rheb1 mutant mice: implications for TORC1-based treatments

    Intact neuronal function in Rheb1 mutant mice: implications for TORC1-based treatments by Goorden, Susanna M I, Abs, Elisabeth, Bruinsma, Caroline F, Riemslagh, Fréderike W, van Woerden, Geeske M, Elgersma, Ype

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  2. 2
    Inducible expression of human C9ORF72 36x G4C2 hexanucleotide repeats is sufficient to cause RAN translation and rapid muscular atrophy in mice

    Inducible expression of human C9ORF72 36x G4C2 hexanucleotide repeats is sufficient to cause RAN translation and rapid muscular atrophy in mice by Riemslagh, F W, van der Toorn, E C, Verhagen, R F M, Maas, A, Bosman, L W J, Hukema, R K, Willemsen, R

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  3. 3
    Inducible expression of human C9ORF72 36x G 4 C 2 hexanucleotide repeats is sufficient to cause RAN translation and rapid muscular atrophy in mice

    Inducible expression of human C9ORF72 36x G 4 C 2 hexanucleotide repeats is sufficient to cause RAN translation and rapid muscular atrophy in mice by Riemslagh, F W, van der Toorn, E C, Verhagen, R F M, Maas, A, Bosman, L W J, Hukema, R K, Willemsen, R

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  4. 4
    Reduction of oxidative stress suppresses poly-GR-mediated toxicity in zebrafish embryos

    Reduction of oxidative stress suppresses poly-GR-mediated toxicity in zebrafish embryos by Riemslagh, Fréderike W, Verhagen, Rob F M, van der Toorn, Esmay C, Smits, Daphne J, Quint, Wim H, van der Linde, Herma C, van Ham, Tjakko J, Willemsen, Rob

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  5. 5
    Inducible expression of human C9ORF72 36× G4C2 hexanucleotide repeats is sufficient to cause RAN translation and rapid muscular atrophy in mice

    Inducible expression of human C9ORF72 36× G4C2 hexanucleotide repeats is sufficient to cause RAN translation and rapid muscular atrophy in mice by Riemslagh, Fréderike W., van der Toorn, Esmay C., Verhagen, Rob F. M., Maas, Alex, Bosman, Laurens W. J., Hukema, Renate K., Willemsen, Rob

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  6. 6
    HR23B pathology preferentially co-localizes with p62, pTDP-43 and poly-GA in C9ORF72-linked frontotemporal dementia and amyotrophic lateral sclerosis

    HR23B pathology preferentially co-localizes with p62, pTDP-43 and poly-GA in C9ORF72-linked frontotemporal dementia and amyotrophic lateral sclerosis by Riemslagh, Frederike W, Lans, Hannes, Seelaar, Harro, Severijnen, Lies-Anne W F M, Melhem, Shamiram, Vermeulen, Wim, Aronica, Eleonora, Pasterkamp, R Jeroen, van Swieten, John C, Willemsen, Rob

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  7. 7
    A new inducible transgenic mouse model for C9orf72-associated GGGGCC repeat expansion supports a gain-of-function mechanism in C9orf72-associated ALS and FTD

    A new inducible transgenic mouse model for C9orf72-associated GGGGCC repeat expansion supports a gain-of-function mechanism in C9orf72-associated ALS and FTD by Hukema, Renate K, Riemslagh, Fréderike W, Melhem, Shamiram, van der Linde, Herma C, Severijnen, Lies-Anne Wfm, Edbauer, Dieter, Maas, Alex, Charlet-Berguerand, Nicolas, Willemsen, Rob, van Swieten, John C

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  8. 8
    Retraction Note to: A new inducible transgenic mouse model for C9orf72-associated GGGGCC repeat expansion supports a gain-of-function mechanism in C9orf72-associated ALS and FTD

    Retraction Note to: A new inducible transgenic mouse model for C9orf72-associated GGGGCC repeat expansion supports a gain-of-function mechanism in C9orf72-associated ALS and FTD by Hukema, Renate K, Riemslagh, Fréderike W, Melhem, Shamiram, van der Linde, Herma C, Severijnen, Lies-Anne W F M, Edbauer, Dieter, Maas, Alex, Charlet-Berguerand, Nicolas, Willemsen, Rob, van Swieten, John C

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