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Increased Afterload Augments Sunitinib-Induced Cardiotoxicity in an Engineered Cardiac Microtissue Model
by
Truitt, Rachel, PhD
,
Mu, Anbin
,
Corbin, Elise A., PhD
,
Vite, Alexia, PhD
,
Brandimarto, Jeffrey, PhD
,
Ky, Bonnie, MD, MSCE
,
Margulies, Kenneth B., MD
Published in
JACC. Basic to translational science
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Jacc. Basic To Translational Science
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Afterload
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Apoptosis
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Cardiac & Cardiovascular Systems
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Cardiotoxicity
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Cardiovascular
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Cardiovascular System & Cardiology
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Life Sciences & Biomedicine
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Preclinical Research
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Science & Technology
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Sunitinib
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Tissue Engineering
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Toxicology
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Tyrosine Kinase Inhibitors
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