SMAD4 Mutations in Colorectal Cancer Probably Occur before Chromosomal Instability, but after Divergence of the Microsatellite Instability Pathway by Woodford-Richens, K. L., Rowan, A. J., Gorman, P., Halford, S., Bicknell, D. C., Wasan, H. S., Roylance, R. R., Bodmer, W. F., Tomlinson, I. P. M.

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An Ancestral Ashkenazi Haplotype at the HMPS/CRAC1 Locus on 15q13–q14 Is Associated with Hereditary Mixed Polyposis Syndrome by Jaeger, E.E.M., Woodford-Richens, K.L., Lockett, M., Rowan, A.J., Sawyer, E.J., Heinimann, K., Rozen, P., Murday, V.A., Whitelaw, S.C., Ginsberg, A., Atkin, W.S., Lynch, H.T., Southey, M.C., Debinski, H., Eng, C., Bodmer, W.F., Talbot, I.C., Hodgson, S.V., Thomas, H.J.W., Tomlinson, I.P.M.

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Allelic variation at the interleukin-1 receptor antagonist gene is associated with early postmenopausal bone loss at the spine by Keen, R.W, Woodford-Richens, K.L, Lanchbury, J.S, Spector, T.D

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CDX2 mutations do not account for juvenile polyposis or Peutz–Jeghers syndrome and occur infrequently in sporadic colorectal cancers by Woodford-Richens, K L, Halford, S, Rowan, A, Bevan, S, Aaltonen, L A, Wasan, H, Bicknell, D, Bodmer, W F, Houlston, R S, Tomlinson, I P M

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Association of polymorphism at the type I collagen (COL1A1) locus with reduced bone mineral density, increased fracture risk, and increased collagen turnover by Keen, R. W., Woodford‐Richens, K. L., Grant, S. F. A., Ralston, S. H., Lanchbury, J. S., Spector, T. D.

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Peak bone mass, early menopausal bone loss and polymorphism at the oestrogen receptor gene by Keen, RW, Woodford-Richens, KL, Lanchbury, JS, Spector, TD

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Germline Mutations in BMPR1A/ALK3 Cause a Subset of Cases of Juvenile Polyposis Syndrome and of Cowden and Bannayan-Riley-Ruvalcaba Syndromes by Zhou, Xiao-Ping, Woodford-Richens, Kelly, Lehtonen, Rainer, Kurose, Keisuke, Aldred, Micheala, Hampel, Heather, Launonen, Virpi, Virta, Sanno, Pilarski, Robert, Salovaara, Reijo, Bodmer, Walter F., Conrad, Beth A., Dunlop, Malcolm, Hodgson, Shirley V., Iwama, Takeo, Järvinen, Heikki, Kellokumpu, Ilmo, Kim, J.C., Leggett, Barbara, Markie, David, Mecklin, Jukka-Pekka, Neale, Kay, Phillips, Robin, Piris, Juan, Rozen, Paul, Houlston, Richard S., Aaltonen, Lauri A., Tomlinson, Ian P.M., Eng, Charis

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Germline mutations in the TGF-β and Wnt signalling pathways are a rare cause of the “multiple” adenoma phenotype by Lipton, L, Sieber, O M, Thomas, H J W, Hodgson, S V, Tomlinson, I P M, Woodford-Richens, K

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Germline mutations in the TGF-ß and Wnt signalling pathways are a rare cause of the "multiple" adenoma phenotype by Lipton, L, Sieber, O, Thomas, H, Hodgson, S, Tomlinson, I, Woodford-Richens, K

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Germline mutations in the TGF-[beta] and Wnt signalling pathways are a rare cause of the "multiple" adenoma phenotype by Lipton, L, Sieber, O M, Thomas, H J W, Hodgson, S V, Tomlinson, I P M, Woodford-Richens, K

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