Acute hydrocephalus following carbon monoxide poisoning

Carbon monoxide remains a significant cause of poisoning in children. Cerebral edema is often the cause of significant morbidity and mortality in exposed children. While lesions of the basal ganglia have been well documented, the advent of neuroimaging has allowed antemortem demonstration of infarct...

Full description

Saved in:
Bibliographic Details
Published in:Pediatric neurology 1997-10, Vol.17 (3), p.270-273
Main Authors: So, Gerald M., Kosofsky, Barry E., Southern, James F.
Format: Article
Language:English
Subjects:
Acute Disease
Biological and medical sciences
Carbon Monoxide Poisoning - complications
Child, Preschool
Domestic and cosmetic products toxicology
Humans
Hydrocephalus - chemically induced
Hydrocephalus - diagnostic imaging
Male
Medical sciences
Tomography, X-Ray Computed
Toxicology
Citations: Items that this one cites
Items that cite this one
Online Access:Get full text
Tags: Add Tag
No Tags, Be the first to tag this record!
Description
Summary:Carbon monoxide remains a significant cause of poisoning in children. Cerebral edema is often the cause of significant morbidity and mortality in exposed children. While lesions of the basal ganglia have been well documented, the advent of neuroimaging has allowed antemortem demonstration of infarctions of the globus pallidus and putamen with carbon monoxide intoxication. Acute hydrocephalus following carbon monoxide poisoning has been a rare occurrence. We report a 2 year 6 month-old boy who, to our knowledge, represents the first reported case in which repeat computed tomography documented the evolution of hydrocephalus due to carbon monoxide exposure in a child.
ISSN:0887-8994
1873-5150
DOI:10.1016/S0887-8994(97)00097-0