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Mechanisms of implant damage annealing and transient enhanced diffusion in Si
Interactions between self-interstitials (I) and {113} interstitial defects during annealing of Si implant damage have been studied. At low damage levels diffusion is ultrafast, driven by I released direct from the ion collision cascade. At higher damage levels, free I are quenched by nucleation of {...
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Published in: | Applied physics letters 1994-12, Vol.65 (23), p.2981-2983 |
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Language: | English |
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cited_by | cdi_FETCH-LOGICAL-c291t-3efe1b3621f94df291f5205e90f6b1c1923906465d604bf21b062e6bf45ddf483 |
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cites | cdi_FETCH-LOGICAL-c291t-3efe1b3621f94df291f5205e90f6b1c1923906465d604bf21b062e6bf45ddf483 |
container_end_page | 2983 |
container_issue | 23 |
container_start_page | 2981 |
container_title | Applied physics letters |
container_volume | 65 |
creator | Cowern, N. E. B. van de Walle, G. F. A. Zalm, P. C. Vandenhoudt, D. W. E. |
description | Interactions between self-interstitials (I) and {113} interstitial defects during annealing of Si implant damage have been studied. At low damage levels diffusion is ultrafast, driven by I released direct from the ion collision cascade. At higher damage levels, free I are quenched by nucleation of {113} defects. We show that the transient enhanced diffusion seen in most previous studies arises from the subsequent dissolution of the {113} defects. |
doi_str_mv | 10.1063/1.112483 |
format | article |
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At higher damage levels, free I are quenched by nucleation of {113} defects. We show that the transient enhanced diffusion seen in most previous studies arises from the subsequent dissolution of the {113} defects.</abstract><doi>10.1063/1.112483</doi><tpages>3</tpages></addata></record> |
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title | Mechanisms of implant damage annealing and transient enhanced diffusion in Si |
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