Galectin‐3 and suppression of tumorigenicity 2: Two emerging cardiac biomarkers that may be predictors of cardiac fibrosis development in sport

According to recent research, ST2 is a new biomarker for predicting heart failure. [...]recent studies have highlighted that repeated bouts of acute, high-intensity exercise lead to deleterious changes in heart tissue that may manifest as an abrupt change in cardiac biomarkers, and ST2 and Gal-3 may...

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Bibliographic Details
Published in:Experimental physiology 2023-10, Vol.108 (10), p.1257-1258
Main Author: Goff, Caroline Le
Format: Article
Language:English
Subjects:
Biomarkers
Biomarkers - analysis
cardiac fibrosis
Cardiovascular & respiratory systems
endurance
Fibrosis
Fibrosis - genetics
Fibrosis - pathology
Galectin 3
Galectin 3 - analysis
Galectin 3 - genetics
Heart
Heart Diseases
Heart Diseases - genetics
Heart Diseases - metabolism
Heart Diseases - pathology
Human health sciences
Humans
IL1RL1 protein, human
Interleukin-1 Receptor-Like 1 Protein
Interleukin-1 Receptor-Like 1 Protein - analysis
Interleukin-1 Receptor-Like 1 Protein - genetics
LGALS3 protein, human
Nutrition and Dietetics
Physiology
Physiology (medical)
Sciences de la santé humaine
sport
Sports
suppression of tumorigenicity 2
Systèmes cardiovasculaire & respiratoire
Tumorigenicity
Viewpoint
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Summary:According to recent research, ST2 is a new biomarker for predicting heart failure. [...]recent studies have highlighted that repeated bouts of acute, high-intensity exercise lead to deleterious changes in heart tissue that may manifest as an abrupt change in cardiac biomarkers, and ST2 and Gal-3 may thus provide significant information (Le Goff et al., 2020). The emergence of novel cardiac biomarkers Gal-3 and ST2 could now provide the opportunity to explore which athletes may be most at risk of experiencing sudden cardiac death (SCD), since cardiac fibrosis could contributed to the development of SCD and Gal-3 and ST2 could give additional information compared with other well-established biomarkers.
ISSN:0958-0670
1469-445X
1469-445X
DOI:10.1113/EP091428