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Experimental encephalitis caused by Taenia crassiceps cysticerci in mice
To present the experimental model of neurocysticercosis (NCC) caused by Taenia crassiceps cysticerci, to describe the inflammatory process, susceptibility, or resistance of BALB/c and C57BL/6 mice to this infection, and to describe the host-parasite relationship. The animals were intracranially inoc...
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Published in: | Arquivos de neuro-psiquiatria 2012-04, Vol.70 (4), p.287-292 |
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creator | Matos-Silva, Hidelberto Reciputti, Bruno Pereira Paula, Elbio Cândido de Oliveira, André Luiz Moura, Vânia Beatriz Lopes Vinaud, Marina Clare Oliveira, Milton Adriano Pelli Lino-Júnior, Ruy de Souza |
description | To present the experimental model of neurocysticercosis (NCC) caused by Taenia crassiceps cysticerci, to describe the inflammatory process, susceptibility, or resistance of BALB/c and C57BL/6 mice to this infection, and to describe the host-parasite relationship.
The animals were intracranially inoculated with initial stage T. crassiceps cysticerci. They were euthanized at 7, 30, 60, and 90 days after the inoculation. Their encephala were removed for the histopathologic analysis, classification of the parasites, and inflammatory lesions.
Experimental NCC was observed on both mice lineages. BALB/c mice presented inflammatory lesions with greater intensity, inducing necrosis on late stage parasites, and with an acute inflammation pattern, while C57BL/6 mice showed greater capability on provoking early necrosis in the cysticerci, which showed a chronic inflammation pattern.
This experimental model induced NCC on mice with characteristic inflammation and lesions. C57BL/6 mice were able to induce precocious necrosis of the parasites presenting inflammatory lesions with lower intensity. |
doi_str_mv | 10.1590/S0004-282X2012005000010 |
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The animals were intracranially inoculated with initial stage T. crassiceps cysticerci. They were euthanized at 7, 30, 60, and 90 days after the inoculation. Their encephala were removed for the histopathologic analysis, classification of the parasites, and inflammatory lesions.
Experimental NCC was observed on both mice lineages. BALB/c mice presented inflammatory lesions with greater intensity, inducing necrosis on late stage parasites, and with an acute inflammation pattern, while C57BL/6 mice showed greater capability on provoking early necrosis in the cysticerci, which showed a chronic inflammation pattern.
This experimental model induced NCC on mice with characteristic inflammation and lesions. C57BL/6 mice were able to induce precocious necrosis of the parasites presenting inflammatory lesions with lower intensity.</description><identifier>ISSN: 0004-282X</identifier><identifier>ISSN: 1678-4227</identifier><identifier>EISSN: 1678-4227</identifier><identifier>DOI: 10.1590/S0004-282X2012005000010</identifier><identifier>PMID: 22358311</identifier><language>eng</language><publisher>Brazil: Academia Brasileira de Neurologia - ABNEURO</publisher><subject>Animal models ; Animals ; BALB/c ; C57BL/6 ; Classification ; Cysticercosis ; Disease Models, Animal ; Encephalitis ; Encephalitis - parasitology ; Encephalitis - pathology ; Female ; Host-Parasite Interactions ; Inflammation ; Inoculation ; Mice ; Mice, Inbred BALB C ; Mice, Inbred C57BL ; Necrosis ; neurocisticercose ; Neurocysticercosis - parasitology ; Neurocysticercosis - pathology ; NEUROSCIENCES ; Parasites ; PSYCHIATRY ; Severity of Illness Index ; Taenia ; Taenia - physiology ; Taenia crassiceps</subject><ispartof>Arquivos de neuro-psiquiatria, 2012-04, Vol.70 (4), p.287-292</ispartof><rights>This work is licensed under a Creative Commons Attribution-NonCommercial 4.0 International License.</rights><lds50>peer_reviewed</lds50><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c500t-7f11e95141fc939a7c4a9b48f6bd69d5fe60ca0ab817a616eb8382366b11db413</citedby><cites>FETCH-LOGICAL-c500t-7f11e95141fc939a7c4a9b48f6bd69d5fe60ca0ab817a616eb8382366b11db413</cites></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><link.rule.ids>230,314,780,784,885,24150,27924,27925</link.rule.ids><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/22358311$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Matos-Silva, Hidelberto</creatorcontrib><creatorcontrib>Reciputti, Bruno Pereira</creatorcontrib><creatorcontrib>Paula, Elbio Cândido de</creatorcontrib><creatorcontrib>Oliveira, André Luiz</creatorcontrib><creatorcontrib>Moura, Vânia Beatriz Lopes</creatorcontrib><creatorcontrib>Vinaud, Marina Clare</creatorcontrib><creatorcontrib>Oliveira, Milton Adriano Pelli</creatorcontrib><creatorcontrib>Lino-Júnior, Ruy de Souza</creatorcontrib><title>Experimental encephalitis caused by Taenia crassiceps cysticerci in mice</title><title>Arquivos de neuro-psiquiatria</title><addtitle>Arq Neuropsiquiatr</addtitle><description>To present the experimental model of neurocysticercosis (NCC) caused by Taenia crassiceps cysticerci, to describe the inflammatory process, susceptibility, or resistance of BALB/c and C57BL/6 mice to this infection, and to describe the host-parasite relationship.
The animals were intracranially inoculated with initial stage T. crassiceps cysticerci. They were euthanized at 7, 30, 60, and 90 days after the inoculation. Their encephala were removed for the histopathologic analysis, classification of the parasites, and inflammatory lesions.
Experimental NCC was observed on both mice lineages. BALB/c mice presented inflammatory lesions with greater intensity, inducing necrosis on late stage parasites, and with an acute inflammation pattern, while C57BL/6 mice showed greater capability on provoking early necrosis in the cysticerci, which showed a chronic inflammation pattern.
This experimental model induced NCC on mice with characteristic inflammation and lesions. C57BL/6 mice were able to induce precocious necrosis of the parasites presenting inflammatory lesions with lower intensity.</description><subject>Animal models</subject><subject>Animals</subject><subject>BALB/c</subject><subject>C57BL/6</subject><subject>Classification</subject><subject>Cysticercosis</subject><subject>Disease Models, Animal</subject><subject>Encephalitis</subject><subject>Encephalitis - parasitology</subject><subject>Encephalitis - pathology</subject><subject>Female</subject><subject>Host-Parasite Interactions</subject><subject>Inflammation</subject><subject>Inoculation</subject><subject>Mice</subject><subject>Mice, Inbred BALB C</subject><subject>Mice, Inbred C57BL</subject><subject>Necrosis</subject><subject>neurocisticercose</subject><subject>Neurocysticercosis - parasitology</subject><subject>Neurocysticercosis - pathology</subject><subject>NEUROSCIENCES</subject><subject>Parasites</subject><subject>PSYCHIATRY</subject><subject>Severity of Illness Index</subject><subject>Taenia</subject><subject>Taenia - physiology</subject><subject>Taenia crassiceps</subject><issn>0004-282X</issn><issn>1678-4227</issn><issn>1678-4227</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2012</creationdate><recordtype>article</recordtype><sourceid>DOA</sourceid><recordid>eNqNUcFu1DAUtCoquhR-AXLkkvKe7djxEVUtrVSJA63EzXp2nOJVNlnsRGL_Hm93WQ5cOPnZnhmPZxj7gHCFjYFP3wBA1rzl3zkgB2jKHhDO2AqVbmvJuX7FVifQBXuT8xqAS2P0a3bBuWhagbhidze_tiHFTRhnGqow-rD9QUOcY648LTl0ldtVjxTGSJVPlHMsiHK3y3OZko9VHKtNGd-y856GHN4d10v2dHvzeH1XP3z9cn_9-aH2xeNc6x4xmAYl9t4IQ9pLMk62vXKdMl3TBwWegFyLmhSq4FrRcqGUQ-ycRHHJ7g-63URruy3WKe3sRNG-HEzp2VIq3oZgoXPBe3JkdC-JpDOhVcJTD-gcb0TRujpoZR_DMNn1tKSxmLcv8dq_8YLcx7snfDwQtmn6uYQ8203MPgwDjWFaskVQBo0Wov0PKHBdgmhkgeoD1Kcp5xT607cQ7L7uf_wc6y7M98dHFrcJ3Yn3p1_xG9dKoq0</recordid><startdate>20120401</startdate><enddate>20120401</enddate><creator>Matos-Silva, Hidelberto</creator><creator>Reciputti, Bruno Pereira</creator><creator>Paula, Elbio Cândido de</creator><creator>Oliveira, André Luiz</creator><creator>Moura, Vânia Beatriz Lopes</creator><creator>Vinaud, Marina Clare</creator><creator>Oliveira, Milton Adriano Pelli</creator><creator>Lino-Júnior, Ruy de Souza</creator><general>Academia Brasileira de Neurologia - ABNEURO</general><general>Academia Brasileira de Neurologia (ABNEURO)</general><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7X8</scope><scope>7TK</scope><scope>GPN</scope><scope>DOA</scope></search><sort><creationdate>20120401</creationdate><title>Experimental encephalitis caused by Taenia crassiceps cysticerci in mice</title><author>Matos-Silva, Hidelberto ; Reciputti, Bruno Pereira ; Paula, Elbio Cândido de ; Oliveira, André Luiz ; Moura, Vânia Beatriz Lopes ; Vinaud, Marina Clare ; Oliveira, Milton Adriano Pelli ; Lino-Júnior, Ruy de Souza</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c500t-7f11e95141fc939a7c4a9b48f6bd69d5fe60ca0ab817a616eb8382366b11db413</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2012</creationdate><topic>Animal models</topic><topic>Animals</topic><topic>BALB/c</topic><topic>C57BL/6</topic><topic>Classification</topic><topic>Cysticercosis</topic><topic>Disease Models, Animal</topic><topic>Encephalitis</topic><topic>Encephalitis - parasitology</topic><topic>Encephalitis - pathology</topic><topic>Female</topic><topic>Host-Parasite Interactions</topic><topic>Inflammation</topic><topic>Inoculation</topic><topic>Mice</topic><topic>Mice, Inbred BALB C</topic><topic>Mice, Inbred C57BL</topic><topic>Necrosis</topic><topic>neurocisticercose</topic><topic>Neurocysticercosis - parasitology</topic><topic>Neurocysticercosis - pathology</topic><topic>NEUROSCIENCES</topic><topic>Parasites</topic><topic>PSYCHIATRY</topic><topic>Severity of Illness Index</topic><topic>Taenia</topic><topic>Taenia - physiology</topic><topic>Taenia crassiceps</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Matos-Silva, Hidelberto</creatorcontrib><creatorcontrib>Reciputti, Bruno Pereira</creatorcontrib><creatorcontrib>Paula, Elbio Cândido de</creatorcontrib><creatorcontrib>Oliveira, André Luiz</creatorcontrib><creatorcontrib>Moura, Vânia Beatriz Lopes</creatorcontrib><creatorcontrib>Vinaud, Marina Clare</creatorcontrib><creatorcontrib>Oliveira, Milton Adriano Pelli</creatorcontrib><creatorcontrib>Lino-Júnior, Ruy de Souza</creatorcontrib><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>MEDLINE - Academic</collection><collection>Neurosciences Abstracts</collection><collection>SciELO</collection><collection>DOAJ Directory of Open Access Journals</collection><jtitle>Arquivos de neuro-psiquiatria</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Matos-Silva, Hidelberto</au><au>Reciputti, Bruno Pereira</au><au>Paula, Elbio Cândido de</au><au>Oliveira, André Luiz</au><au>Moura, Vânia Beatriz Lopes</au><au>Vinaud, Marina Clare</au><au>Oliveira, Milton Adriano Pelli</au><au>Lino-Júnior, Ruy de Souza</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Experimental encephalitis caused by Taenia crassiceps cysticerci in mice</atitle><jtitle>Arquivos de neuro-psiquiatria</jtitle><addtitle>Arq Neuropsiquiatr</addtitle><date>2012-04-01</date><risdate>2012</risdate><volume>70</volume><issue>4</issue><spage>287</spage><epage>292</epage><pages>287-292</pages><issn>0004-282X</issn><issn>1678-4227</issn><eissn>1678-4227</eissn><abstract>To present the experimental model of neurocysticercosis (NCC) caused by Taenia crassiceps cysticerci, to describe the inflammatory process, susceptibility, or resistance of BALB/c and C57BL/6 mice to this infection, and to describe the host-parasite relationship.
The animals were intracranially inoculated with initial stage T. crassiceps cysticerci. They were euthanized at 7, 30, 60, and 90 days after the inoculation. Their encephala were removed for the histopathologic analysis, classification of the parasites, and inflammatory lesions.
Experimental NCC was observed on both mice lineages. BALB/c mice presented inflammatory lesions with greater intensity, inducing necrosis on late stage parasites, and with an acute inflammation pattern, while C57BL/6 mice showed greater capability on provoking early necrosis in the cysticerci, which showed a chronic inflammation pattern.
This experimental model induced NCC on mice with characteristic inflammation and lesions. C57BL/6 mice were able to induce precocious necrosis of the parasites presenting inflammatory lesions with lower intensity.</abstract><cop>Brazil</cop><pub>Academia Brasileira de Neurologia - ABNEURO</pub><pmid>22358311</pmid><doi>10.1590/S0004-282X2012005000010</doi><tpages>6</tpages><oa>free_for_read</oa></addata></record> |
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subjects | Animal models Animals BALB/c C57BL/6 Classification Cysticercosis Disease Models, Animal Encephalitis Encephalitis - parasitology Encephalitis - pathology Female Host-Parasite Interactions Inflammation Inoculation Mice Mice, Inbred BALB C Mice, Inbred C57BL Necrosis neurocisticercose Neurocysticercosis - parasitology Neurocysticercosis - pathology NEUROSCIENCES Parasites PSYCHIATRY Severity of Illness Index Taenia Taenia - physiology Taenia crassiceps |
title | Experimental encephalitis caused by Taenia crassiceps cysticerci in mice |
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