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HIV-Associated Tuberculosis 2012
K. A. Wilkinson 1,2 and M. E. Torok 3,4 and S. Schwander 5 and G. Meintjes 2 1, National Institute for Medical Research, Mill Hill, London NW7 1AA, UK 2, Institute of Infectious Disease and Molecular Medicine, University of Cape Town, Rondebosch, Cape Town 7701, South Africa 3, Department of Infecti...
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Published in: | Clinical & developmental immunology 2012-01, Vol.2012 (2012), p.1-2 |
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Main Authors: | , , , |
Format: | Article |
Language: | English |
Subjects: | |
Citations: | Items that cite this one |
Online Access: | Get full text |
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Summary: | K. A. Wilkinson 1,2 and M. E. Torok 3,4 and S. Schwander 5 and G. Meintjes 2 1, National Institute for Medical Research, Mill Hill, London NW7 1AA, UK 2, Institute of Infectious Disease and Molecular Medicine, University of Cape Town, Rondebosch, Cape Town 7701, South Africa 3, Department of Infectious Diseases, Addenbrooke's Hospital, Cambridge CB2 0QQ, UK 4, Department of Medicine, Addenbrooke's Hospital, University of Cambridge, Box 157, Cambridge CB2 0QQ, UK 5, Department of Environmental and Occupational Health, Center for Global Public Health, School of Public Health, UMDNJ, NJ, USA Received 31 May 2012; Accepted 31 May 2012 This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Chronic HIV infection leads to excessive production of proinflammatory cytokines which leads to the generation of free radicals which are in turn scavenged by free glutathione. [...]the excessive production of free radicals in HIV-infected individuals or reduced glutathione synthesis may lead to the depletion of glutathione. |
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ISSN: | 1740-2522 2314-8861 1740-2530 2314-7156 |
DOI: | 10.1155/2012/950964 |