Interaction between adipocytes and high-density lipoprotein:new insights into the mechanism of obesity-induced dyslipidemia and atherosclerosis

Obesity is the most common nutritional disorder worldwide and is associated with dyslipidemia and atherosclerotic cardiovascular disease. The hallmark of dyslipidemia in obesity is low high density lipoprotein (HDL) cholesterol (HDL-C) levels. Moreover, the quality of HDL is also changed in the obes...

Full description

Saved in:
Bibliographic Details
Published in:Lipids in health and disease 2019-12, Vol.18 (1), p.223-223, Article 223
Main Authors: Zhang, Tianhua, Chen, Jin, Tang, Xiaoyu, Luo, Qin, Xu, Danyan, Yu, Bilian
Format: Article
Language:English
Subjects:
Abdomen
Adipocyte
Adipocytes
Adipocytes - physiology
Adipose tissue
Adipose Tissue - physiopathology
Animals
Apolipoprotein A-I - physiology
Arteriosclerosis
Atherosclerosis
Atherosclerosis - etiology
Atherosclerosis - physiopathology
Atherosclerosis - prevention & control
Body fat
Cardiovascular disease
Cardiovascular diseases
Cholesterol
Cholesterol, HDL - blood
Cytokines
Dyslipidemia
Dyslipidemias - etiology
Dyslipidemias - physiopathology
Energy
Energy management
Energy storage
Fatty acids
Female
HDL
High density lipoprotein
Hormones
Humans
Insulin resistance
Lipids
Lipolysis
Lipoproteins
Lipoproteins, HDL - physiology
Liver
Male
Metabolic disorders
Metabolism
Obesity
Obesity - complications
Physiological aspects
Proteins
Review
Thermogenesis
Triglycerides
Tumor necrosis factor-TNF
Type 2 diabetes
Citations: Items that this one cites
Items that cite this one
Online Access:Get full text
Tags: Add Tag
No Tags, Be the first to tag this record!
cited_by cdi_FETCH-LOGICAL-c560t-5e319c96952fece48d3a733f170084d257600bbacbb46ae46f27277c8e682d603
cites cdi_FETCH-LOGICAL-c560t-5e319c96952fece48d3a733f170084d257600bbacbb46ae46f27277c8e682d603
container_end_page 223
container_issue 1
container_start_page 223
container_title Lipids in health and disease
container_volume 18
creator Zhang, Tianhua
Chen, Jin
Tang, Xiaoyu
Luo, Qin
Xu, Danyan
Yu, Bilian
description Obesity is the most common nutritional disorder worldwide and is associated with dyslipidemia and atherosclerotic cardiovascular disease. The hallmark of dyslipidemia in obesity is low high density lipoprotein (HDL) cholesterol (HDL-C) levels. Moreover, the quality of HDL is also changed in the obese setting. However, there are still some disputes on the explanations for this phenomenon. There is increasing evidence that adipose tissue, as an energy storage tissue, participates in several metabolism activities, such as hormone secretion and cholesterol efflux. It can influence overall reverse cholesterol transport and plasma HDL-C level. In obesity individuals, the changes in morphology and function of adipose tissue affect plasma HDL-C levels and HDL function, thus, adipose tissue should be the main target for the treatment of HDL metabolism in obesity. In this review, we will summarize the cross-talk between adipocytes and HDL related to cardiovascular disease and focus on the new insights of the potential mechanism underlying obesity and HDL dysfunction.
doi_str_mv 10.1186/s12944-019-1170-9
format article
fullrecord <record><control><sourceid>gale_doaj_</sourceid><recordid>TN_cdi_doaj_primary_oai_doaj_org_article_965049318c224323ba66b505b4a833de</recordid><sourceformat>XML</sourceformat><sourcesystem>PC</sourcesystem><galeid>A610309837</galeid><doaj_id>oai_doaj_org_article_965049318c224323ba66b505b4a833de</doaj_id><sourcerecordid>A610309837</sourcerecordid><originalsourceid>FETCH-LOGICAL-c560t-5e319c96952fece48d3a733f170084d257600bbacbb46ae46f27277c8e682d603</originalsourceid><addsrcrecordid>eNptkl2L1DAUhoso7rr6A7yRgjfedM1X08QLYVn8GFjwRsG7kCan0wxtMiaZXeZX-JdNd9ZhRyTQhJP3fZqcvFX1GqNLjAV_nzCRjDUIywbjDjXySXWOWcebFuOfTx-tz6oXKW0QIqjj_Hl1RrFgRAh2Xv1e-QxRm-yCr3vIdwC-1tZtg9lnSLX2th7demws-OTyvp7K1jaGDM5_8HBXu1JejzmVRQ51HqGewYzauzTXYahDD4utcd7uDNja7lMhOAuz0_dwXSwxJDMtX5deVs8GPSV49TBfVD8-f_p-_bW5-fZldX1105iWo9y0QLE0ksuWDGCACUt1R-lQuoAEs6TtOEJ9r03fM66B8YF0pOuMAC6I5YheVKsD1wa9UdvoZh33Kmin7gshrpWO2ZVjKclbxGRpmSGEUUJ7zXnforZnWlBqobA-HljbXT-DNeBz1NMJ9HTHu1Gtw63iElOERQG8ewDE8GsHKavZJQPTpD2EXVKEkk4ygjtSpG__kW7CLvrSqkUlREvacqijaq3LBZwfQvmvWaDqimNEkRS0K6rL_6jKWF7HBA-DK_UTAz4YTHmrFGE43hEjtSRSHRKpSiLVkkgli-fN4-YcHX8jSP8A93fdyA</addsrcrecordid><sourcetype>Open Website</sourcetype><iscdi>true</iscdi><recordtype>article</recordtype><pqid>2328852583</pqid></control><display><type>article</type><title>Interaction between adipocytes and high-density lipoprotein:new insights into the mechanism of obesity-induced dyslipidemia and atherosclerosis</title><source>Open Access: PubMed Central</source><source>Publicly Available Content Database</source><creator>Zhang, Tianhua ; Chen, Jin ; Tang, Xiaoyu ; Luo, Qin ; Xu, Danyan ; Yu, Bilian</creator><creatorcontrib>Zhang, Tianhua ; Chen, Jin ; Tang, Xiaoyu ; Luo, Qin ; Xu, Danyan ; Yu, Bilian</creatorcontrib><description>Obesity is the most common nutritional disorder worldwide and is associated with dyslipidemia and atherosclerotic cardiovascular disease. The hallmark of dyslipidemia in obesity is low high density lipoprotein (HDL) cholesterol (HDL-C) levels. Moreover, the quality of HDL is also changed in the obese setting. However, there are still some disputes on the explanations for this phenomenon. There is increasing evidence that adipose tissue, as an energy storage tissue, participates in several metabolism activities, such as hormone secretion and cholesterol efflux. It can influence overall reverse cholesterol transport and plasma HDL-C level. In obesity individuals, the changes in morphology and function of adipose tissue affect plasma HDL-C levels and HDL function, thus, adipose tissue should be the main target for the treatment of HDL metabolism in obesity. In this review, we will summarize the cross-talk between adipocytes and HDL related to cardiovascular disease and focus on the new insights of the potential mechanism underlying obesity and HDL dysfunction.</description><identifier>ISSN: 1476-511X</identifier><identifier>EISSN: 1476-511X</identifier><identifier>DOI: 10.1186/s12944-019-1170-9</identifier><identifier>PMID: 31842884</identifier><language>eng</language><publisher>England: BioMed Central Ltd</publisher><subject>Abdomen ; Adipocyte ; Adipocytes ; Adipocytes - physiology ; Adipose tissue ; Adipose Tissue - physiopathology ; Animals ; Apolipoprotein A-I - physiology ; Arteriosclerosis ; Atherosclerosis ; Atherosclerosis - etiology ; Atherosclerosis - physiopathology ; Atherosclerosis - prevention &amp; control ; Body fat ; Cardiovascular disease ; Cardiovascular diseases ; Cholesterol ; Cholesterol, HDL - blood ; Cytokines ; Dyslipidemia ; Dyslipidemias - etiology ; Dyslipidemias - physiopathology ; Energy ; Energy management ; Energy storage ; Fatty acids ; Female ; HDL ; High density lipoprotein ; Hormones ; Humans ; Insulin resistance ; Lipids ; Lipolysis ; Lipoproteins ; Lipoproteins, HDL - physiology ; Liver ; Male ; Metabolic disorders ; Metabolism ; Obesity ; Obesity - complications ; Physiological aspects ; Proteins ; Review ; Thermogenesis ; Triglycerides ; Tumor necrosis factor-TNF ; Type 2 diabetes</subject><ispartof>Lipids in health and disease, 2019-12, Vol.18 (1), p.223-223, Article 223</ispartof><rights>COPYRIGHT 2019 BioMed Central Ltd.</rights><rights>2019. This work is licensed under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License.</rights><rights>The Author(s). 2019</rights><lds50>peer_reviewed</lds50><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c560t-5e319c96952fece48d3a733f170084d257600bbacbb46ae46f27277c8e682d603</citedby><cites>FETCH-LOGICAL-c560t-5e319c96952fece48d3a733f170084d257600bbacbb46ae46f27277c8e682d603</cites><orcidid>0000-0002-5701-5487</orcidid></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><linktopdf>$$Uhttps://www.ncbi.nlm.nih.gov/pmc/articles/PMC6913018/pdf/$$EPDF$$P50$$Gpubmedcentral$$Hfree_for_read</linktopdf><linktohtml>$$Uhttps://www.proquest.com/docview/2328852583?pq-origsite=primo$$EHTML$$P50$$Gproquest$$Hfree_for_read</linktohtml><link.rule.ids>230,314,725,778,782,883,25736,27907,27908,36995,36996,44573,53774,53776</link.rule.ids><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/31842884$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Zhang, Tianhua</creatorcontrib><creatorcontrib>Chen, Jin</creatorcontrib><creatorcontrib>Tang, Xiaoyu</creatorcontrib><creatorcontrib>Luo, Qin</creatorcontrib><creatorcontrib>Xu, Danyan</creatorcontrib><creatorcontrib>Yu, Bilian</creatorcontrib><title>Interaction between adipocytes and high-density lipoprotein:new insights into the mechanism of obesity-induced dyslipidemia and atherosclerosis</title><title>Lipids in health and disease</title><addtitle>Lipids Health Dis</addtitle><description>Obesity is the most common nutritional disorder worldwide and is associated with dyslipidemia and atherosclerotic cardiovascular disease. The hallmark of dyslipidemia in obesity is low high density lipoprotein (HDL) cholesterol (HDL-C) levels. Moreover, the quality of HDL is also changed in the obese setting. However, there are still some disputes on the explanations for this phenomenon. There is increasing evidence that adipose tissue, as an energy storage tissue, participates in several metabolism activities, such as hormone secretion and cholesterol efflux. It can influence overall reverse cholesterol transport and plasma HDL-C level. In obesity individuals, the changes in morphology and function of adipose tissue affect plasma HDL-C levels and HDL function, thus, adipose tissue should be the main target for the treatment of HDL metabolism in obesity. In this review, we will summarize the cross-talk between adipocytes and HDL related to cardiovascular disease and focus on the new insights of the potential mechanism underlying obesity and HDL dysfunction.</description><subject>Abdomen</subject><subject>Adipocyte</subject><subject>Adipocytes</subject><subject>Adipocytes - physiology</subject><subject>Adipose tissue</subject><subject>Adipose Tissue - physiopathology</subject><subject>Animals</subject><subject>Apolipoprotein A-I - physiology</subject><subject>Arteriosclerosis</subject><subject>Atherosclerosis</subject><subject>Atherosclerosis - etiology</subject><subject>Atherosclerosis - physiopathology</subject><subject>Atherosclerosis - prevention &amp; control</subject><subject>Body fat</subject><subject>Cardiovascular disease</subject><subject>Cardiovascular diseases</subject><subject>Cholesterol</subject><subject>Cholesterol, HDL - blood</subject><subject>Cytokines</subject><subject>Dyslipidemia</subject><subject>Dyslipidemias - etiology</subject><subject>Dyslipidemias - physiopathology</subject><subject>Energy</subject><subject>Energy management</subject><subject>Energy storage</subject><subject>Fatty acids</subject><subject>Female</subject><subject>HDL</subject><subject>High density lipoprotein</subject><subject>Hormones</subject><subject>Humans</subject><subject>Insulin resistance</subject><subject>Lipids</subject><subject>Lipolysis</subject><subject>Lipoproteins</subject><subject>Lipoproteins, HDL - physiology</subject><subject>Liver</subject><subject>Male</subject><subject>Metabolic disorders</subject><subject>Metabolism</subject><subject>Obesity</subject><subject>Obesity - complications</subject><subject>Physiological aspects</subject><subject>Proteins</subject><subject>Review</subject><subject>Thermogenesis</subject><subject>Triglycerides</subject><subject>Tumor necrosis factor-TNF</subject><subject>Type 2 diabetes</subject><issn>1476-511X</issn><issn>1476-511X</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2019</creationdate><recordtype>article</recordtype><sourceid>PIMPY</sourceid><sourceid>DOA</sourceid><recordid>eNptkl2L1DAUhoso7rr6A7yRgjfedM1X08QLYVn8GFjwRsG7kCan0wxtMiaZXeZX-JdNd9ZhRyTQhJP3fZqcvFX1GqNLjAV_nzCRjDUIywbjDjXySXWOWcebFuOfTx-tz6oXKW0QIqjj_Hl1RrFgRAh2Xv1e-QxRm-yCr3vIdwC-1tZtg9lnSLX2th7demws-OTyvp7K1jaGDM5_8HBXu1JejzmVRQ51HqGewYzauzTXYahDD4utcd7uDNja7lMhOAuz0_dwXSwxJDMtX5deVs8GPSV49TBfVD8-f_p-_bW5-fZldX1105iWo9y0QLE0ksuWDGCACUt1R-lQuoAEs6TtOEJ9r03fM66B8YF0pOuMAC6I5YheVKsD1wa9UdvoZh33Kmin7gshrpWO2ZVjKclbxGRpmSGEUUJ7zXnforZnWlBqobA-HljbXT-DNeBz1NMJ9HTHu1Gtw63iElOERQG8ewDE8GsHKavZJQPTpD2EXVKEkk4ygjtSpG__kW7CLvrSqkUlREvacqijaq3LBZwfQvmvWaDqimNEkRS0K6rL_6jKWF7HBA-DK_UTAz4YTHmrFGE43hEjtSRSHRKpSiLVkkgli-fN4-YcHX8jSP8A93fdyA</recordid><startdate>20191216</startdate><enddate>20191216</enddate><creator>Zhang, Tianhua</creator><creator>Chen, Jin</creator><creator>Tang, Xiaoyu</creator><creator>Luo, Qin</creator><creator>Xu, Danyan</creator><creator>Yu, Bilian</creator><general>BioMed Central Ltd</general><general>BioMed Central</general><general>BMC</general><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>3V.</scope><scope>7X7</scope><scope>7XB</scope><scope>88E</scope><scope>8FD</scope><scope>8FE</scope><scope>8FH</scope><scope>8FI</scope><scope>8FJ</scope><scope>8FK</scope><scope>ABUWG</scope><scope>AFKRA</scope><scope>AZQEC</scope><scope>BBNVY</scope><scope>BENPR</scope><scope>BHPHI</scope><scope>CCPQU</scope><scope>DWQXO</scope><scope>FR3</scope><scope>FYUFA</scope><scope>GHDGH</scope><scope>GNUQQ</scope><scope>HCIFZ</scope><scope>K9.</scope><scope>LK8</scope><scope>M0S</scope><scope>M1P</scope><scope>M7P</scope><scope>P64</scope><scope>PIMPY</scope><scope>PQEST</scope><scope>PQQKQ</scope><scope>PQUKI</scope><scope>PRINS</scope><scope>RC3</scope><scope>7X8</scope><scope>5PM</scope><scope>DOA</scope><orcidid>https://orcid.org/0000-0002-5701-5487</orcidid></search><sort><creationdate>20191216</creationdate><title>Interaction between adipocytes and high-density lipoprotein:new insights into the mechanism of obesity-induced dyslipidemia and atherosclerosis</title><author>Zhang, Tianhua ; Chen, Jin ; Tang, Xiaoyu ; Luo, Qin ; Xu, Danyan ; Yu, Bilian</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c560t-5e319c96952fece48d3a733f170084d257600bbacbb46ae46f27277c8e682d603</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2019</creationdate><topic>Abdomen</topic><topic>Adipocyte</topic><topic>Adipocytes</topic><topic>Adipocytes - physiology</topic><topic>Adipose tissue</topic><topic>Adipose Tissue - physiopathology</topic><topic>Animals</topic><topic>Apolipoprotein A-I - physiology</topic><topic>Arteriosclerosis</topic><topic>Atherosclerosis</topic><topic>Atherosclerosis - etiology</topic><topic>Atherosclerosis - physiopathology</topic><topic>Atherosclerosis - prevention &amp; control</topic><topic>Body fat</topic><topic>Cardiovascular disease</topic><topic>Cardiovascular diseases</topic><topic>Cholesterol</topic><topic>Cholesterol, HDL - blood</topic><topic>Cytokines</topic><topic>Dyslipidemia</topic><topic>Dyslipidemias - etiology</topic><topic>Dyslipidemias - physiopathology</topic><topic>Energy</topic><topic>Energy management</topic><topic>Energy storage</topic><topic>Fatty acids</topic><topic>Female</topic><topic>HDL</topic><topic>High density lipoprotein</topic><topic>Hormones</topic><topic>Humans</topic><topic>Insulin resistance</topic><topic>Lipids</topic><topic>Lipolysis</topic><topic>Lipoproteins</topic><topic>Lipoproteins, HDL - physiology</topic><topic>Liver</topic><topic>Male</topic><topic>Metabolic disorders</topic><topic>Metabolism</topic><topic>Obesity</topic><topic>Obesity - complications</topic><topic>Physiological aspects</topic><topic>Proteins</topic><topic>Review</topic><topic>Thermogenesis</topic><topic>Triglycerides</topic><topic>Tumor necrosis factor-TNF</topic><topic>Type 2 diabetes</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Zhang, Tianhua</creatorcontrib><creatorcontrib>Chen, Jin</creatorcontrib><creatorcontrib>Tang, Xiaoyu</creatorcontrib><creatorcontrib>Luo, Qin</creatorcontrib><creatorcontrib>Xu, Danyan</creatorcontrib><creatorcontrib>Yu, Bilian</creatorcontrib><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>ProQuest Central (Corporate)</collection><collection>ProQuest Health &amp; Medical Collection</collection><collection>ProQuest Central (purchase pre-March 2016)</collection><collection>Medical Database (Alumni Edition)</collection><collection>Technology Research Database</collection><collection>ProQuest SciTech Collection</collection><collection>ProQuest Natural Science Collection</collection><collection>Hospital Premium Collection</collection><collection>Hospital Premium Collection (Alumni Edition)</collection><collection>ProQuest Central (Alumni) (purchase pre-March 2016)</collection><collection>ProQuest Central (Alumni)</collection><collection>ProQuest Central</collection><collection>ProQuest Central Essentials</collection><collection>Biological Science Collection</collection><collection>AUTh Library subscriptions: ProQuest Central</collection><collection>ProQuest Natural Science Collection</collection><collection>ProQuest One Community College</collection><collection>ProQuest Central</collection><collection>Engineering Research Database</collection><collection>Health Research Premium Collection</collection><collection>Health Research Premium Collection (Alumni)</collection><collection>ProQuest Central Student</collection><collection>SciTech Premium Collection</collection><collection>ProQuest Health &amp; Medical Complete (Alumni)</collection><collection>ProQuest Biological Science Collection</collection><collection>Health &amp; Medical Collection (Alumni Edition)</collection><collection>Medical Database</collection><collection>ProQuest Biological Science Journals</collection><collection>Biotechnology and BioEngineering Abstracts</collection><collection>Publicly Available Content Database</collection><collection>ProQuest One Academic Eastern Edition (DO NOT USE)</collection><collection>ProQuest One Academic</collection><collection>ProQuest One Academic UKI Edition</collection><collection>ProQuest Central China</collection><collection>Genetics Abstracts</collection><collection>MEDLINE - Academic</collection><collection>PubMed Central (Full Participant titles)</collection><collection>Open Access: DOAJ - Directory of Open Access Journals</collection><jtitle>Lipids in health and disease</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Zhang, Tianhua</au><au>Chen, Jin</au><au>Tang, Xiaoyu</au><au>Luo, Qin</au><au>Xu, Danyan</au><au>Yu, Bilian</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Interaction between adipocytes and high-density lipoprotein:new insights into the mechanism of obesity-induced dyslipidemia and atherosclerosis</atitle><jtitle>Lipids in health and disease</jtitle><addtitle>Lipids Health Dis</addtitle><date>2019-12-16</date><risdate>2019</risdate><volume>18</volume><issue>1</issue><spage>223</spage><epage>223</epage><pages>223-223</pages><artnum>223</artnum><issn>1476-511X</issn><eissn>1476-511X</eissn><abstract>Obesity is the most common nutritional disorder worldwide and is associated with dyslipidemia and atherosclerotic cardiovascular disease. The hallmark of dyslipidemia in obesity is low high density lipoprotein (HDL) cholesterol (HDL-C) levels. Moreover, the quality of HDL is also changed in the obese setting. However, there are still some disputes on the explanations for this phenomenon. There is increasing evidence that adipose tissue, as an energy storage tissue, participates in several metabolism activities, such as hormone secretion and cholesterol efflux. It can influence overall reverse cholesterol transport and plasma HDL-C level. In obesity individuals, the changes in morphology and function of adipose tissue affect plasma HDL-C levels and HDL function, thus, adipose tissue should be the main target for the treatment of HDL metabolism in obesity. In this review, we will summarize the cross-talk between adipocytes and HDL related to cardiovascular disease and focus on the new insights of the potential mechanism underlying obesity and HDL dysfunction.</abstract><cop>England</cop><pub>BioMed Central Ltd</pub><pmid>31842884</pmid><doi>10.1186/s12944-019-1170-9</doi><tpages>1</tpages><orcidid>https://orcid.org/0000-0002-5701-5487</orcidid><oa>free_for_read</oa></addata></record>
fulltext fulltext
identifier ISSN: 1476-511X
ispartof Lipids in health and disease, 2019-12, Vol.18 (1), p.223-223, Article 223
issn 1476-511X
1476-511X
language eng
recordid cdi_doaj_primary_oai_doaj_org_article_965049318c224323ba66b505b4a833de
source Open Access: PubMed Central; Publicly Available Content Database
subjects Abdomen
Adipocyte
Adipocytes
Adipocytes - physiology
Adipose tissue
Adipose Tissue - physiopathology
Animals
Apolipoprotein A-I - physiology
Arteriosclerosis
Atherosclerosis
Atherosclerosis - etiology
Atherosclerosis - physiopathology
Atherosclerosis - prevention & control
Body fat
Cardiovascular disease
Cardiovascular diseases
Cholesterol
Cholesterol, HDL - blood
Cytokines
Dyslipidemia
Dyslipidemias - etiology
Dyslipidemias - physiopathology
Energy
Energy management
Energy storage
Fatty acids
Female
HDL
High density lipoprotein
Hormones
Humans
Insulin resistance
Lipids
Lipolysis
Lipoproteins
Lipoproteins, HDL - physiology
Liver
Male
Metabolic disorders
Metabolism
Obesity
Obesity - complications
Physiological aspects
Proteins
Review
Thermogenesis
Triglycerides
Tumor necrosis factor-TNF
Type 2 diabetes
title Interaction between adipocytes and high-density lipoprotein:new insights into the mechanism of obesity-induced dyslipidemia and atherosclerosis
url http://sfxeu10.hosted.exlibrisgroup.com/loughborough?ctx_ver=Z39.88-2004&ctx_enc=info:ofi/enc:UTF-8&ctx_tim=2025-01-17T01%3A11%3A39IST&url_ver=Z39.88-2004&url_ctx_fmt=infofi/fmt:kev:mtx:ctx&rfr_id=info:sid/primo.exlibrisgroup.com:primo3-Article-gale_doaj_&rft_val_fmt=info:ofi/fmt:kev:mtx:journal&rft.genre=article&rft.atitle=Interaction%20between%20adipocytes%20and%20high-density%20lipoprotein:new%20insights%20into%20the%20mechanism%20of%20obesity-induced%20dyslipidemia%20and%20atherosclerosis&rft.jtitle=Lipids%20in%20health%20and%20disease&rft.au=Zhang,%20Tianhua&rft.date=2019-12-16&rft.volume=18&rft.issue=1&rft.spage=223&rft.epage=223&rft.pages=223-223&rft.artnum=223&rft.issn=1476-511X&rft.eissn=1476-511X&rft_id=info:doi/10.1186/s12944-019-1170-9&rft_dat=%3Cgale_doaj_%3EA610309837%3C/gale_doaj_%3E%3Cgrp_id%3Ecdi_FETCH-LOGICAL-c560t-5e319c96952fece48d3a733f170084d257600bbacbb46ae46f27277c8e682d603%3C/grp_id%3E%3Coa%3E%3C/oa%3E%3Curl%3E%3C/url%3E&rft_id=info:oai/&rft_pqid=2328852583&rft_id=info:pmid/31842884&rft_galeid=A610309837&rfr_iscdi=true