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General Catalytic Deficiency of Hepatitis C Virus RNA Polymerase with an S282T Mutation and Mutually Exclusive Resistance towards 2′-Modified Nucleotide Analogues
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| Published in: | Antimicrobial Agents and Chemotherapy 2006-12, Vol.50 (12), p.4161-4169 |
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| Main Authors: | , , , |
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| cites | cdi_FETCH-LOGICAL-a581t-49d1c9ff2238e7aedea1189798def2134bfb9eab0912515a8908d8cce749ea813 |
| container_end_page | 4169 |
| container_issue | 12 |
| container_start_page | 4161 |
| container_title | Antimicrobial Agents and Chemotherapy |
| container_volume | 50 |
| creator | DUTARTRE, Hélène BUSSETTA, Cécile BORETTO, Joëlle CANARD, Bruno |
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| doi_str_mv | 10.1128/AAC.00433-06 |
| format | article |
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1752 N Street N.W. • Washington DC 20036
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Print ISSN:
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</abstract><cop>Washington, DC</cop><pub>American Society for Microbiology</pub><pmid>17000745</pmid><doi>10.1128/AAC.00433-06</doi><tpages>9</tpages><orcidid>https://orcid.org/0000-0003-4924-1991</orcidid><oa>free_for_read</oa></addata></record> |
| fulltext | fulltext |
| identifier | ISSN: 0066-4804 |
| ispartof | Antimicrobial Agents and Chemotherapy, 2006-12, Vol.50 (12), p.4161-4169 |
| issn | 0066-4804 1098-6596 |
| language | eng |
| recordid | cdi_highwire_asm_aac_50_12_4161 |
| source | American Society for Microbiology; PubMed Central |
| subjects | Antibiotics. Antiinfectious agents. Antiparasitic agents Antiviral Agents Binding Sites Biological and medical sciences Cytidine - analogs & derivatives Cytidine - genetics Drug Resistance, Viral Hepacivirus Hepacivirus - enzymology Hepatitis C virus Human immunodeficiency virus Human viral diseases Humans Infectious diseases Kinetics Life Sciences Medical sciences Microbiology and Parasitology Models, Molecular Mutation Nucleotides Nucleotides - genetics Pharmacology. Drug treatments RNA Replicase RNA Replicase - genetics RNA, Viral RNA, Viral - analysis RNA, Viral - biosynthesis Viral diseases Viral hepatitis Virology |
| title | General Catalytic Deficiency of Hepatitis C Virus RNA Polymerase with an S282T Mutation and Mutually Exclusive Resistance towards 2′-Modified Nucleotide Analogues |
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