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Specific β2AR blocker ICI 118,551 actively decreases contraction through a Gi-Coupled form of the β2AR in myocytes from failing human heart

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Published in:Circulation (New York, N.Y.) N.Y.), 2002-05, Vol.105 (21), p.2497-2503
Main Authors: GONG, Haibin, HONG SUN, KOCH, Walter J, RAU, Thomas, ESCHENHAGEN, Thomas, RAVENS, Ursula, HEUBACH, Jürgen F, ADAMSON, Dawn L, HARDING, Sian E
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Language:eng ; jpn
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container_end_page 2503
container_issue 21
container_start_page 2497
container_title Circulation (New York, N.Y.)
container_volume 105
creator GONG, Haibin
HONG SUN
KOCH, Walter J
RAU, Thomas
ESCHENHAGEN, Thomas
RAVENS, Ursula
HEUBACH, Jürgen F
ADAMSON, Dawn L
HARDING, Sian E
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doi_str_mv 10.1161/01.cir.0000017187.61348.95
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ispartof Circulation (New York, N.Y.), 2002-05, Vol.105 (21), p.2497-2503
issn 0009-7322
1524-4539
language eng ; jpn
recordid cdi_pascalfrancis_primary_14185200
source EZB Electronic Journals Library
subjects Biological and medical sciences
Cardiology. Vascular system
Heart
Heart failure, cardiogenic pulmonary edema, cardiac enlargement
Medical sciences
title Specific β2AR blocker ICI 118,551 actively decreases contraction through a Gi-Coupled form of the β2AR in myocytes from failing human heart
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