Inhibition of Vascular Endothelial Abl1 Signaling Protects Against Food-Induced Anaphylaxis in Mice

Methods We employed BALB/c wild-type (WT), intestinal IL-9Tg and VE-specific Abl1 kinase-deficient (Cdh5CreAbl1fl/fl) mice and pharmacologic inhibitors (Imatinib) and models of anaphylaxis to determine the requirement of Abl1 kinase in IgE-mediated reactions.

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Published in:Journal of allergy and clinical immunology 2017-02, Vol.139 (2), p.AB277-AB277
Main Authors: Hogan, Simon P., PhD, Waggoner, Lisa, MS, Wu, David, PhD, Yamani, Amnah N., MS
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Language:English
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Allergy and Immunology
Rodents
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container_title Journal of allergy and clinical immunology
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creator Hogan, Simon P., PhD
Waggoner, Lisa, MS
Wu, David, PhD
Yamani, Amnah N., MS
description Methods We employed BALB/c wild-type (WT), intestinal IL-9Tg and VE-specific Abl1 kinase-deficient (Cdh5CreAbl1fl/fl) mice and pharmacologic inhibitors (Imatinib) and models of anaphylaxis to determine the requirement of Abl1 kinase in IgE-mediated reactions.
doi_str_mv 10.1016/j.jaci.2016.12.890
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title Inhibition of Vascular Endothelial Abl1 Signaling Protects Against Food-Induced Anaphylaxis in Mice
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