Muscle metaboreflex control of cardiac output and peripheral vasoconstriction exhibit different latencies

Experiments were designed to determine 1) the mechanisms mediating metaboreflex-induced increases in systemic arterial pressure (SAP) in response to total vascular occlusion of hindlimb blood flow [e.g., increases in cardiac output (CO) vs. peripheral vasoconstriction] and 2) whether the individual...

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Bibliographic Details
Published in:American journal of physiology. Heart and circulatory physiology 2000-02, Vol.47 (2), p.H530-H537
Main Authors: AUGUSTYNIAK, R. A, ANSORGE, E. J, O'LEARY, D. S
Format: Article
Language:English
Subjects:
Biological and medical sciences
Blood pressure
Blood vessels
Circulatory system
Fundamental and applied biological sciences. Psychology
Heart
Metabolism
Muscular system
Striated muscle. Tendons
Vertebrates: osteoarticular system, musculoskeletal system
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Summary:Experiments were designed to determine 1) the mechanisms mediating metaboreflex-induced increases in systemic arterial pressure (SAP) in response to total vascular occlusion of hindlimb blood flow [e.g., increases in cardiac output (CO) vs. peripheral vasoconstriction] and 2) whether the individual mechanisms display differential latencies for the onset of the responses. Responses were observed in seven dogs performing steady-state treadmill exercise of mild and moderate workloads (3.2 km/h at 0% grade and 6.4 km/h at 10% grade).
ISSN:0363-6135
1522-1539