Malathion exposure induces the endocrine disruption and growth retardation in the catfish, Clarias batrachus (Linn.)

Highlights ► Organophosphorous pesticide retard somatic as well as gonadal growth of fish. ► Malathion disrupts the secretory patterns of T, E2, T4, T3, GH and IGF-I. ► It also diminishes food consumption by affecting the olfactory sensation. ► Chronic malathion exposure causes lipolysis to meet the...

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Bibliographic Details
Published in:General and comparative endocrinology 2013-01, Vol.181 (15), p.139-145
Main Authors: Lal, Bechan, Sarang, Mukesh Kumar, Kumar, Pankaj
Format: Article
Language:English
Subjects:
animal growth
Animals
body weight
catfish
Catfishes
Clarias batrachus
Eating - drug effects
Endocrine Disruptors - toxicity
Endocrinology & Metabolism
Estradiol
Estradiol - metabolism
feeding behavior
Fish growth
Food consumption
food intake
freshwater
Growth Hormone - metabolism
growth retardation
IGF-I
insulin-like growth factor I
Insulin-Like Growth Factor I - metabolism
lipolysis
Lipolysis - drug effects
liver
Malathion
Malathion - toxicity
muscles
protein content
sensation
somatotropin
Testosterone
Testosterone - metabolism
triiodothyronine
Triiodothyronine - metabolism
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Summary:Highlights ► Organophosphorous pesticide retard somatic as well as gonadal growth of fish. ► Malathion disrupts the secretory patterns of T, E2, T4, T3, GH and IGF-I. ► It also diminishes food consumption by affecting the olfactory sensation. ► Chronic malathion exposure causes lipolysis to meet the stressful condition.
ISSN:0016-6480
1095-6840
DOI:10.1016/j.ygcen.2012.11.004