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New immunologic pathways in the pathogenesis of leprosy: Role for Th22 cytokines in the polar forms of the disease

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Published in:Journal of the American Academy of Dermatology 2015-04, Vol.72 (4), p.729-730
Main Authors: de Lima Silveira, Edvaldo, MSc, de Sousa, Jorge Rodrigues, BSc, de Sousa Aarão, Tinara Leila, PhD, Fuzii, Hellen Thais, PhD, Dias, Leonidas Braga, PhD, Carneiro, Francisca Regina Oliveira, PhD, Quaresma, Juarez Antonio Simões, PhD
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subjects Adult
Dermatology
Female
Fibroblast Growth Factor 2 - analysis
Fibroblast Growth Factor 2 - physiology
Host-Pathogen Interactions - immunology
Humans
Immune Evasion
Interleukin-13 - analysis
Interleukin-13 - physiology
Interleukin-22
Interleukins - analysis
Interleukins - physiology
Leprosy, Lepromatous - etiology
Leprosy, Lepromatous - immunology
Leprosy, Lepromatous - pathology
Leprosy, Tuberculoid - etiology
Leprosy, Tuberculoid - immunology
Leprosy, Tuberculoid - pathology
Macrophages - microbiology
Male
Mycobacterium leprae - immunology
T-Lymphocytes, Helper-Inducer - chemistry
T-Lymphocytes, Helper-Inducer - classification
T-Lymphocytes, Helper-Inducer - immunology
Tumor Necrosis Factor-alpha - analysis
Tumor Necrosis Factor-alpha - physiology
title New immunologic pathways in the pathogenesis of leprosy: Role for Th22 cytokines in the polar forms of the disease
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