HPRT and glycophorin A mutations in foundry workers: relationship to PAH exposure and to PAH-DNA adducts

Mutations were evaluated in workers in an iron foundry with exposure to polycyclic aromatic hydrocarbons (PAHs), measured by personal and area monitoring, ranging from

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Bibliographic Details
Published in:Carcinogenesis (New York) 1993-05, Vol.14 (5), p.969-973
Main Authors: Perera, F.P., Tang, D.L., O'Neill, J.P., Bigbee, W.L., Albertini, R.J., Santella, R., Ottman, R., Tsai, W.Y., Dickey, C., Mooney, L.A., Savela, K., Hemminki, K.
Format: Article
Language:English
Subjects:
adducts
Adult
Biological and medical sciences
Cohort Studies
DNA
DNA - blood
exposure
Female
Glycophorin - genetics
glycophorin A
Host-tumor relations. Immunology. Biological markers
Humans
hypoxanthine phosphoribosyl transferase
Hypoxanthine Phosphoribosyltransferase - genetics
Iron
Leukocytes - metabolism
Male
man
Medical sciences
Middle Aged
Mutagenesis
mutation
Occupational Exposure
polycyclic aromatic hydrocarbons
Polycyclic Compounds - blood
Tumors
Citations: Items that cite this one
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Description
Summary:Mutations were evaluated in workers in an iron foundry with exposure to polycyclic aromatic hydrocarbons (PAHs), measured by personal and area monitoring, ranging from
ISSN:0143-3334
1460-2180
DOI:10.1093/carcin/14.5.969