Role of MEK, PI3, p38, Tyrosine, and mTOR Kinases in Regulation of Heart Resistance to the Arrhythmogenic Action of Short-Term Ischemia and Reperfusion

MEK, PI3, p38, tyrosine, and mTOR kinases are not involved in the regulation of heart resistance to the arrhythmogenic action of short-term ischemia/reperfusion in non-adapted rats.

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Bibliographic Details
Published in:Bulletin of experimental biology and medicine 2015-04, Vol.158 (6), p.729-731
Main Authors: Maslov, L. N., Mukhomedzyanov, A. V., Tsibulnikov, S. Yu, Chauski, E. I., Khaliulin, I. G., Portnichenko, A. G.
Format: Article
Language:English
Subjects:
Animals
Arrhythmia
Arrhythmias, Cardiac - enzymology
Arrhythmias, Cardiac - etiology
Biomedical and Life Sciences
Biomedicine
Cell Biology
Flavonoids - pharmacology
Imidazoles - pharmacology
Internal Medicine
Ischemia
Laboratory Medicine
MAP Kinase Kinase Kinases - antagonists & inhibitors
MAP Kinase Kinase Kinases - metabolism
Myocardial Reperfusion Injury - complications
Myocardial Reperfusion Injury - enzymology
p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors
p38 Mitogen-Activated Protein Kinases - metabolism
Pathology
Phenols
Phosphatidylinositol 3-Kinases - antagonists & inhibitors
Phosphatidylinositol 3-Kinases - metabolism
Phosphotransferases
Pyridines - pharmacology
Rats
Rats, Wistar
Sirolimus - pharmacology
TOR Serine-Threonine Kinases - antagonists & inhibitors
TOR Serine-Threonine Kinases - metabolism
Tyrosine
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Description
Summary:MEK, PI3, p38, tyrosine, and mTOR kinases are not involved in the regulation of heart resistance to the arrhythmogenic action of short-term ischemia/reperfusion in non-adapted rats.
ISSN:0007-4888
1573-8221
DOI:10.1007/s10517-015-2848-2