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Iron overload triggers mitochondrial fragmentation via calcineurin-sensitive signals in HT-22 hippocampal neuron cells

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Published in:Toxicology (Amsterdam) 2015-11, Vol.337, p.39-46
Main Authors: Park, Junghyung, Lee, Dong Gil, Kim, Bokyung, Park, Sun-Ji, Kim, Jung-Hak, Lee, Sang-Rae, Chang, Kyu-Tae, Lee, Hyun-Shik, Lee, Dong-Seok
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container_issue
container_start_page 39
container_title Toxicology (Amsterdam)
container_volume 337
creator Park, Junghyung
Lee, Dong Gil
Kim, Bokyung
Park, Sun-Ji
Kim, Jung-Hak
Lee, Sang-Rae
Chang, Kyu-Tae
Lee, Hyun-Shik
Lee, Dong-Seok
description Graphical abstract
doi_str_mv 10.1016/j.tox.2015.08.009
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source ScienceDirect Journals
subjects Activation
Animals
Apoptosis - drug effects
Calcines
Calcineurin
Calcineurin - drug effects
Calcineurin - pharmacology
Cell Line
Cell Survival - drug effects
Drp1
Dynamics
Dynamins - drug effects
Emergency
Fragmentation
Hippocampus - drug effects
Hippocampus - pathology
Iron
Iron overload
Iron Overload - pathology
Mice
Mitochondria - drug effects
Mitochondria - pathology
Mitochondria - ultrastructure
Mitochondrial dynamics
Neurons
Neurons - drug effects
Neurons - pathology
Neurotoxicity
Phosphorylation
RNA Interference
Signal Transduction - drug effects
Toxicity
title Iron overload triggers mitochondrial fragmentation via calcineurin-sensitive signals in HT-22 hippocampal neuron cells
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