Accelerated acidosis in response to variable fetal heart rate decelerations in chronically hypoxic ovine fetuses

Background Due to limitations of technology, clinicians are typically unable to determine if human fetuses are normoxic or moderately, chronically hypoxic. Risk factors for chronic hypoxia include fetal growth restriction, which is associated with an increased incidence of oligohydramnios and thus a...

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Bibliographic Details
Published in:American journal of obstetrics and gynecology 2016-02, Vol.214 (2), p.270.e1-270.e8
Main Authors: Amaya, Kevin E., DO, Matushewski, Brad, Durosier, L. Daniel, MD, Frasch, Martin G., MD, PhD, Richardson, Bryan S., MD, Ross, Michael G., MD, MPH
Format: Article
Language:English
Subjects:
acidosis
Acidosis - metabolism
Animals
asphyxia
Chronic Disease
Female
Fetal Hypoxia - metabolism
Fetus - metabolism
Heart Rate, Fetal
Hydrogen-Ion Concentration
hypoxia
Labor, Obstetric
Models, Animal
Obstetrics and Gynecology
Pregnancy
Sheep
Umbilical Cord - blood supply
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Summary:Background Due to limitations of technology, clinicians are typically unable to determine if human fetuses are normoxic or moderately, chronically hypoxic. Risk factors for chronic hypoxia include fetal growth restriction, which is associated with an increased incidence of oligohydramnios and thus a risk for umbilical cord occlusion (UCO) and variable fetal heart rate (FHR) decelerations. At delivery, fetal growth restriction infants (
ISSN:0002-9378
1097-6868
DOI:10.1016/j.ajog.2015.09.084