Fatal unintentional occupational poisonings by hydrofluoric acid in the U.S

• Published in: American journal of industrial medicine 2001-08, Vol.40 (2), p.215-220
• Main Authors: Blodgett, David W., Suruda, Anthony J., Crouch, Barbara Insley
• Format: Article
• Language: English
• Subjects: Adult; Biological and medical sciences; calcium chloride; Chemical and industrial products toxicology. Toxic occupational diseases; chemical poisoning; fatalities; gluconate; Humans; hydrofluoric acid; Hydrofluoric Acid - poisoning; Male; Medical sciences; Middle Aged; Occupational Exposure - adverse effects; poisoning; Poisoning - mortality; Toxicology; United States - epidemiology; Various organic compounds; workplace fatality; workplace poisoning
• ISSN: 0271-3586; 1097-0274
• DOI: 10.1002/ajim.1090

Background Case reports have identified hydrofluoric acid (HF) as causing fatal work injury, and HF has both local and systemic toxicity. Surveillance for HF‐related mortality is problematic because of the lack of unique coding for this acid in hospital records and vital statistics. Methods We identified HF‐related fatal work injuries investigated by the Occupational Safety and Health Administration (OSHA) for 1984–94 from coding of Hazardous Substance 1460 (HF) and requested case investigation files under the Freedom of Information Act. We attempted to identify HF‐related deaths in the US for the same period through literature case reports, the Consumer Product Safety Commission, and the American Association of Poison Control Centers (AAPCC). Results For the 11 year period, OSHA investigated nine deaths in eight incidents which involved HF. Four deaths were from skin contact with concentrated HF, and five deaths involved both skin contact and inhalation of vapor. Unsafe work practices were factors in all of the deaths. Calcium chloride or gluconate was noted to have been administered to five of the nine victims. Calcium was administered 90 min after exposure to two victims, and more than 6 h after exposure to a third. We were able to establish that the regional poison control center had been contacted in regard to only one victim. For the period 1984–94, we were able to identify no additional deaths from CPSC reports, one additional death from AAPCC annual reports, and four other deaths from case reports in the medical literature. Conclusions For the period of this study, OSHA records identified the greatest number of HF‐related fatalities. The limited information in the records suggest that some victims did not receive appropriate medical care, nor was the regional poison center contacted regarding care. The full extent of health problems related to HF could be better quantified if usual surveillance sources, such as vital records, included unique coding for this acid. Am. J. Ind. Med. 40:215–220, 2001. © 2001 Wiley‐Liss, Inc.