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Erythrocytes from hereditary xerocytosis patients heterozygous for KCNN4 V282M exhibit increased spontaneous Gardos channel‐like activity inhibited by senicapoc

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Bibliographic Details
Published in:	American journal of hematology 2017-06, Vol.92 (6), p.E108-E110
Main Authors:	Rivera, Alicia, Vandorpe, David H., Shmukler, Boris E., Gallagher, Denis R., Fikry, Christopher C., Kuypers, Frans A., Brugnara, Carlo, Snyder, L. Michael, Alper, Seth L.
Format:	Article
Language:	English
Subjects:	Acetamides - pharmacology Acetamides - therapeutic use Anemia, Hemolytic, Congenital - blood Anemia, Hemolytic, Congenital - genetics Cell Count Drug Evaluation, Preclinical Erythrocyte Deformability Erythrocytes Erythrocytes - drug effects Erythrocytes - metabolism Gain of Function Mutation Hematology Heterozygote Humans Hydrops Fetalis - blood Hydrops Fetalis - genetics In Vitro Techniques Intermediate-Conductance Calcium-Activated Potassium Channels - antagonists & inhibitors Intermediate-Conductance Calcium-Activated Potassium Channels - genetics Ion Transport - drug effects Mutation, Missense Osmotic Fragility Patch-Clamp Techniques Potassium - blood Potassium Channel Blockers - pharmacology Potassium Channel Blockers - therapeutic use Potassium channels (calcium-gated) Trityl Compounds - pharmacology Trityl Compounds - therapeutic use
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