Active decompression and distraction sugosteogenesis for the treatment of calcifying odontogenic cyst

The calcifying odontogenic cyst is as a benign, rare developmental odontogenic cyst with a wide range of histologic characteristics. It may present along with other odontogenic pathologies such as odontoma, ameloblastoma, adenomatoid odontogenic tumor, ameloblastic fibroma, and ameloblastic fibro-od...

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Bibliographic Details
Published in:Oral and maxillofacial surgery 2021-03, Vol.25 (1), p.89-97
Main Authors: Moreno-Rodríguez, Pedro, Guerrero, Lidia M., Gómez-Delgado, Andrés, Castro-Núñez, Jaime
Format: Article
Language:English
Subjects:
Ameloblastoma - diagnostic imaging
Ameloblastoma - surgery
Biopsy
Case Report
Cysts
Decompression
Dentistry
Fractures
General anesthesia
Humans
Maxillofacial surgery
Medicine
Medicine & Public Health
Odontogenic Cyst, Calcifying - diagnostic imaging
Odontogenic Cyst, Calcifying - surgery
Odontogenic Tumors
Odontoma
Oral and Maxillofacial Surgery
Pathology
Teeth
Tumors
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Summary:The calcifying odontogenic cyst is as a benign, rare developmental odontogenic cyst with a wide range of histologic characteristics. It may present along with other odontogenic pathologies such as odontoma, ameloblastoma, adenomatoid odontogenic tumor, ameloblastic fibroma, and ameloblastic fibro-odontoma. Clinically, it can be an either intra- or extraosseous painless swelling that can produce cortical expansion. It affects mostly the anterior area of the mandible. Radiographically, it appears as a well-circumscribed unilocular radiolucency containing flecks of indistinct radiopacities. In about one third of cases, an impacted tooth is associated. In this paper, we employ a patient with an enormous calcifying odontogenic cyst to review both the pathology and active decompression and distraction sugosteogenesis, a novel technique employed to treat odontogenic entities. This dual approach usually results in an accelerated bone healing (sugosteogenesis), partial removal of the cystic epithelium, thickening of the wall, and migration of chronic inflammatory cells which triggered epithelial modulation, ultimately causing a realignment in the biologic behavior of the lesion.
ISSN:1865-1550
1865-1569
DOI:10.1007/s10006-020-00885-0