Loading…
Unlikely partners in weight loss?
The classic role of bile acids is as a key component of cholesterol homeostasis. New evidence recently published in Nature ( Watanabe et al., 2006), however, describes a bile acid signaling pathway that controls energy expenditure through induction of thyroid hormone signaling pathways. This work ma...
Saved in:
| Published in: | Cell metabolism 2006-02, Vol.3 (2), p.81-82 |
|---|---|
| Main Author: | |
| Format: | Article |
| Language: | English |
| Subjects: | |
| Citations: | Items that this one cites Items that cite this one |
| Online Access: | Get full text |
| Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
| cited_by | cdi_FETCH-LOGICAL-c398t-7c4b43a601827353c74e3e9b02c8da57f12ddfba0d91f7a53ddeeebe14e93f1e3 |
|---|---|
| cites | cdi_FETCH-LOGICAL-c398t-7c4b43a601827353c74e3e9b02c8da57f12ddfba0d91f7a53ddeeebe14e93f1e3 |
| container_end_page | 82 |
| container_issue | 2 |
| container_start_page | 81 |
| container_title | Cell metabolism |
| container_volume | 3 |
| creator | Wondisford, Fredric E. |
| description | The classic role of bile acids is as a key component of cholesterol homeostasis. New evidence recently published in
Nature (
Watanabe et al., 2006), however, describes a bile acid signaling pathway that controls energy expenditure through induction of thyroid hormone signaling pathways. This work may point to new approaches to tissue-specific metabolic regulation. |
| doi_str_mv | 10.1016/j.cmet.2006.01.008 |
| format | article |
| fullrecord | <record><control><sourceid>proquest_cross</sourceid><recordid>TN_cdi_proquest_miscellaneous_67628645</recordid><sourceformat>XML</sourceformat><sourcesystem>PC</sourcesystem><els_id>S1550413106000325</els_id><sourcerecordid>67628645</sourcerecordid><originalsourceid>FETCH-LOGICAL-c398t-7c4b43a601827353c74e3e9b02c8da57f12ddfba0d91f7a53ddeeebe14e93f1e3</originalsourceid><addsrcrecordid>eNp9kE1LAzEQhoMoVqt_wIOsF2-7ziT7CYJI8QsKXuw5ZJNZTd2PmmyV_ntTWvDmaebwvC8zD2MXCAkC5jfLRHc0JhwgTwATgPKAnWAleFykHA7DnmUQpyhwwk69XwKIXFTimE0wT7NKQHnCrhZ9az-p3UQr5caenI9sH_2Qff8Yo3bw_u6MHTWq9XS-n1O2eHx4mz3H89enl9n9PNaiKse40GmdCpUDlrwQmdBFSoKqGrgujcqKBrkxTa3AVNgUKhPGEFFNmFIlGiQxZde73pUbvtbkR9lZr6ltVU_D2su8yHkZ7g4g34HahfscNXLlbKfcRiLIrRi5lFsxcitGAsogJoQu9-3ruiPzF9mbCMDtDqDw47clJ7221Gsy1pEepRnsf_2_f2xznw</addsrcrecordid><sourcetype>Aggregation Database</sourcetype><iscdi>true</iscdi><recordtype>article</recordtype><pqid>67628645</pqid></control><display><type>article</type><title>Unlikely partners in weight loss?</title><source>BACON - Elsevier - GLOBAL_SCIENCEDIRECT-OPENACCESS</source><creator>Wondisford, Fredric E.</creator><creatorcontrib>Wondisford, Fredric E.</creatorcontrib><description>The classic role of bile acids is as a key component of cholesterol homeostasis. New evidence recently published in
Nature (
Watanabe et al., 2006), however, describes a bile acid signaling pathway that controls energy expenditure through induction of thyroid hormone signaling pathways. This work may point to new approaches to tissue-specific metabolic regulation.</description><identifier>ISSN: 1550-4131</identifier><identifier>EISSN: 1932-7420</identifier><identifier>DOI: 10.1016/j.cmet.2006.01.008</identifier><identifier>PMID: 16459308</identifier><language>eng</language><publisher>United States: Elsevier Inc</publisher><subject>Adipose Tissue, Brown - metabolism ; Animals ; Bile Acids and Salts - metabolism ; Cholesterol - blood ; Cholesterol - metabolism ; Energy Metabolism - physiology ; Homeostasis - physiology ; Humans ; Mice ; Models, Biological ; Signal Transduction - physiology ; Thyroid Hormones - metabolism ; Weight Loss - physiology</subject><ispartof>Cell metabolism, 2006-02, Vol.3 (2), p.81-82</ispartof><rights>2006 Elsevier Inc.</rights><lds50>peer_reviewed</lds50><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c398t-7c4b43a601827353c74e3e9b02c8da57f12ddfba0d91f7a53ddeeebe14e93f1e3</citedby><cites>FETCH-LOGICAL-c398t-7c4b43a601827353c74e3e9b02c8da57f12ddfba0d91f7a53ddeeebe14e93f1e3</cites></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><link.rule.ids>314,780,784,27924,27925</link.rule.ids><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/16459308$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Wondisford, Fredric E.</creatorcontrib><title>Unlikely partners in weight loss?</title><title>Cell metabolism</title><addtitle>Cell Metab</addtitle><description>The classic role of bile acids is as a key component of cholesterol homeostasis. New evidence recently published in
Nature (
Watanabe et al., 2006), however, describes a bile acid signaling pathway that controls energy expenditure through induction of thyroid hormone signaling pathways. This work may point to new approaches to tissue-specific metabolic regulation.</description><subject>Adipose Tissue, Brown - metabolism</subject><subject>Animals</subject><subject>Bile Acids and Salts - metabolism</subject><subject>Cholesterol - blood</subject><subject>Cholesterol - metabolism</subject><subject>Energy Metabolism - physiology</subject><subject>Homeostasis - physiology</subject><subject>Humans</subject><subject>Mice</subject><subject>Models, Biological</subject><subject>Signal Transduction - physiology</subject><subject>Thyroid Hormones - metabolism</subject><subject>Weight Loss - physiology</subject><issn>1550-4131</issn><issn>1932-7420</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2006</creationdate><recordtype>article</recordtype><recordid>eNp9kE1LAzEQhoMoVqt_wIOsF2-7ziT7CYJI8QsKXuw5ZJNZTd2PmmyV_ntTWvDmaebwvC8zD2MXCAkC5jfLRHc0JhwgTwATgPKAnWAleFykHA7DnmUQpyhwwk69XwKIXFTimE0wT7NKQHnCrhZ9az-p3UQr5caenI9sH_2Qff8Yo3bw_u6MHTWq9XS-n1O2eHx4mz3H89enl9n9PNaiKse40GmdCpUDlrwQmdBFSoKqGrgujcqKBrkxTa3AVNgUKhPGEFFNmFIlGiQxZde73pUbvtbkR9lZr6ltVU_D2su8yHkZ7g4g34HahfscNXLlbKfcRiLIrRi5lFsxcitGAsogJoQu9-3ruiPzF9mbCMDtDqDw47clJ7221Gsy1pEepRnsf_2_f2xznw</recordid><startdate>20060201</startdate><enddate>20060201</enddate><creator>Wondisford, Fredric E.</creator><general>Elsevier Inc</general><scope>6I.</scope><scope>AAFTH</scope><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7X8</scope></search><sort><creationdate>20060201</creationdate><title>Unlikely partners in weight loss?</title><author>Wondisford, Fredric E.</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c398t-7c4b43a601827353c74e3e9b02c8da57f12ddfba0d91f7a53ddeeebe14e93f1e3</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2006</creationdate><topic>Adipose Tissue, Brown - metabolism</topic><topic>Animals</topic><topic>Bile Acids and Salts - metabolism</topic><topic>Cholesterol - blood</topic><topic>Cholesterol - metabolism</topic><topic>Energy Metabolism - physiology</topic><topic>Homeostasis - physiology</topic><topic>Humans</topic><topic>Mice</topic><topic>Models, Biological</topic><topic>Signal Transduction - physiology</topic><topic>Thyroid Hormones - metabolism</topic><topic>Weight Loss - physiology</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Wondisford, Fredric E.</creatorcontrib><collection>ScienceDirect Open Access Titles</collection><collection>Elsevier:ScienceDirect:Open Access</collection><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>MEDLINE - Academic</collection><jtitle>Cell metabolism</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Wondisford, Fredric E.</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Unlikely partners in weight loss?</atitle><jtitle>Cell metabolism</jtitle><addtitle>Cell Metab</addtitle><date>2006-02-01</date><risdate>2006</risdate><volume>3</volume><issue>2</issue><spage>81</spage><epage>82</epage><pages>81-82</pages><issn>1550-4131</issn><eissn>1932-7420</eissn><abstract>The classic role of bile acids is as a key component of cholesterol homeostasis. New evidence recently published in
Nature (
Watanabe et al., 2006), however, describes a bile acid signaling pathway that controls energy expenditure through induction of thyroid hormone signaling pathways. This work may point to new approaches to tissue-specific metabolic regulation.</abstract><cop>United States</cop><pub>Elsevier Inc</pub><pmid>16459308</pmid><doi>10.1016/j.cmet.2006.01.008</doi><tpages>2</tpages><oa>free_for_read</oa></addata></record> |
| fulltext | fulltext |
| identifier | ISSN: 1550-4131 |
| ispartof | Cell metabolism, 2006-02, Vol.3 (2), p.81-82 |
| issn | 1550-4131 1932-7420 |
| language | eng |
| recordid | cdi_proquest_miscellaneous_67628645 |
| source | BACON - Elsevier - GLOBAL_SCIENCEDIRECT-OPENACCESS |
| subjects | Adipose Tissue, Brown - metabolism Animals Bile Acids and Salts - metabolism Cholesterol - blood Cholesterol - metabolism Energy Metabolism - physiology Homeostasis - physiology Humans Mice Models, Biological Signal Transduction - physiology Thyroid Hormones - metabolism Weight Loss - physiology |
| title | Unlikely partners in weight loss? |
| url | http://sfxeu10.hosted.exlibrisgroup.com/loughborough?ctx_ver=Z39.88-2004&ctx_enc=info:ofi/enc:UTF-8&ctx_tim=2025-01-03T04%3A06%3A23IST&url_ver=Z39.88-2004&url_ctx_fmt=infofi/fmt:kev:mtx:ctx&rfr_id=info:sid/primo.exlibrisgroup.com:primo3-Article-proquest_cross&rft_val_fmt=info:ofi/fmt:kev:mtx:journal&rft.genre=article&rft.atitle=Unlikely%20partners%20in%20weight%20loss?&rft.jtitle=Cell%20metabolism&rft.au=Wondisford,%20Fredric%20E.&rft.date=2006-02-01&rft.volume=3&rft.issue=2&rft.spage=81&rft.epage=82&rft.pages=81-82&rft.issn=1550-4131&rft.eissn=1932-7420&rft_id=info:doi/10.1016/j.cmet.2006.01.008&rft_dat=%3Cproquest_cross%3E67628645%3C/proquest_cross%3E%3Cgrp_id%3Ecdi_FETCH-LOGICAL-c398t-7c4b43a601827353c74e3e9b02c8da57f12ddfba0d91f7a53ddeeebe14e93f1e3%3C/grp_id%3E%3Coa%3E%3C/oa%3E%3Curl%3E%3C/url%3E&rft_id=info:oai/&rft_pqid=67628645&rft_id=info:pmid/16459308&rfr_iscdi=true |