The combination of shared family environment and individual-specific developmental deviance as a cause for treated psychiatric morbidity in children

Objective:  There is an incomplete understanding of why some children growing up in the same family are in need of treatment for psychiatric morbidity whilst their siblings are not. The present paper examined the possible role of individual‐specific developmental risk factors. Method:  Three case–co...

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Bibliographic Details
Published in:Acta psychiatrica Scandinavica 2005-11, Vol.112 (5), p.376-384
Main Authors: Gunther, N., Drukker, M., Feron, F., Korebrits, A., van Os, J.
Format: Article
Language:English
Subjects:
Adolescent
Adult
Biological and medical sciences
Case-Control Studies
Child
Child clinical studies
Child Development
Child psychology
Child, Preschool
community mental health services
Environment
Family Relations
Female
Humans
Male
Medical sciences
Mental Disorders - etiology
Mental Health
Miscellaneous
Motor Skills Disorders
Psychology. Psychoanalysis. Psychiatry
Psychopathology. Psychiatry
Risk Factors
Siblings
Speech Disorders
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Summary:Objective:  There is an incomplete understanding of why some children growing up in the same family are in need of treatment for psychiatric morbidity whilst their siblings are not. The present paper examined the possible role of individual‐specific developmental risk factors. Method:  Three case–control analyses were conducted: i) 80 children referred to the Community Mental Health Centre (cases) and 320 population controls, ii) 68 healthy siblings of cases and 272 population controls, and iii) 80 children and 68 healthy siblings. Measures of development and psychosocial circumstances were obtained from routine, longitudinal, standardized child medical records. Results:  Given shared family environments, additional presence of delays in speech and motor development contributed most to differential sibling mental health outcomes. In addition, cases displayed both earlier expression and more severe levels of developmental behavioural deviance than their healthy siblings, who in turn had higher levels of behavioural deviance than population controls. Conclusion:  In siblings sharing a familial risk environment, development of psychiatric morbidity may be canalized through additional individual‐specific developmental exposures.
ISSN:0001-690X
1600-0447
DOI:10.1111/j.1600-0447.2005.00563.x