Possible Dual Role of Ubiquitin-Proteasome System in the Atherosclerotic Plaque Progression

(1) presented atherosclerosis as a protein-quality disease caused by the accumulation at the vascular level of unfolded/misfolded proteins linking ubiquitin-proteasome system (UPS) activity to atherosclerotic plaque formation.

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Bibliographic Details
Published in:Journal of the American College of Cardiology 2008-10, Vol.52 (16), p.1350-1351
Main Authors: Di Filippo, Clara, PhD, Marfella, Raffaele, MD, D'Amico, Michele, PhD
Format: Article
Language:English
Subjects:
Atherosclerosis
Atherosclerosis - enzymology
Atherosclerosis - pathology
Atherosclerosis - physiopathology
Biomarkers - analysis
Cardiology
Cardiovascular
Disease Progression
Endothelium
Humans
Internal Medicine
Medical research
Prognosis
Proteasome Endopeptidase Complex - metabolism
Proteasome Endopeptidase Complex - physiology
Proteins
Sensitivity and Specificity
Ubiquitin-Protein Ligase Complexes - metabolism
Ubiquitin-Protein Ligase Complexes - physiology
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Description
Summary:(1) presented atherosclerosis as a protein-quality disease caused by the accumulation at the vascular level of unfolded/misfolded proteins linking ubiquitin-proteasome system (UPS) activity to atherosclerotic plaque formation.
ISSN:0735-1097
1558-3597
DOI:10.1016/j.jacc.2008.06.045