Loss of mRor1 Enhances the Heart and Skeletal Abnormalities in mRor2-Deficient Mice: Redundant and Pleiotropic Functions of mRor1 and mRor2 Receptor Tyrosine Kinases

• Published in: Molecular and Cellular Biology 2001-12, Vol.21 (24), p.8329-8335
• Main Authors: Nomi, Masashi, Oishi, Isao, Kani, Shuichi, Suzuki, Hiroaki, Matsuda, Takeru, Yoda, Akinori, Kitamura, Makiko, Itoh, Kyoko, Takeuchi, Shigeto, Takeda, Kiyoshi, Akira, Shizuo, Ikeya, Makoto, Takada, Shinji, Minami, Yasuhiro
• Format: Article
• Language: English
• Subjects: Animals; Animals, Newborn; Bone and Bones - abnormalities; Bone and Bones - metabolism; Heart Defects, Congenital - genetics; In Situ Hybridization; Lung - metabolism; Mammalian Genetic Models with Minimal or Complex Phenotypes; Mice; Mice, Inbred C57BL; Mice, Transgenic; Models, Genetic; mRor1 gene; mRor2 gene; Mutation; Phenotype; Protein Structure, Tertiary; Receptor Protein-Tyrosine Kinases; Receptor Tyrosine Kinase-like Orphan Receptors; Receptors, Cell Surface - genetics; Receptors, Cell Surface - physiology; Time Factors
• ISSN: 0270-7306; 1098-5549; 1098-5549
• DOI: 10.1128/MCB.21.24.8329-8335.2001

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