Neural and metabolic control of arteriolar diameter

During sympathetic nerve activity, sympathetic transmitter causes depolarizations which trigger voltage dependent Ca++ entry. The ability of arteriolar membranes to sum together successive e.j.ps is changed as a consequence of cellular activity, i.e. increased [K+]o. Furthermore, the ability of arte...

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Bibliographic Details
Published in:Japanese journal of pharmacology 1992, Vol.58, p.174-178
Main Authors: Hirst, G.D.S., Edwards, F.R., Silverberg, G.D.
Format: Article
Language:English
Subjects:
Animals
Arterioles - anatomy & histology
Arterioles - innervation
Arterioles - metabolism
Biological and medical sciences
Fundamental and applied biological sciences. Psychology
Humans
Neurons - physiology
Striated muscle. Tendons
Sympathetic Nervous System - physiology
Vertebrates: osteoarticular system, musculoskeletal system
Citations: Items that this one cites
Online Access:Get full text
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Summary:During sympathetic nerve activity, sympathetic transmitter causes depolarizations which trigger voltage dependent Ca++ entry. The ability of arteriolar membranes to sum together successive e.j.ps is changed as a consequence of cellular activity, i.e. increased [K+]o. Furthermore, the ability of arteriolar membrane to permit voltage dependent Ca++ entry is modified by a second consequence of cellular activity, i.e. changes in external pH.
ISSN:0021-5198
1347-3506
DOI:10.1016/S0021-5198(19)59910-3