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Arterio-venous shunts or low oxygen utilization?
An idea of arteriovenous shunts (AVS) was proposed for explanation of dynamic regulation of oxygenation and venous hyperoxia. A formula enabling calculation of AVS and real CO 2 production has recently been derived by comparing data of arterial and venous blood gases. Regarding venous hyperoxia, the...
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Published in: | Human & experimental toxicology 2010-02, Vol.29 (2), p.141-144 |
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description | An idea of arteriovenous shunts (AVS) was proposed for explanation of dynamic regulation of oxygenation and venous hyperoxia. A formula enabling calculation of AVS and real CO 2 production has recently been derived by comparing data of arterial and venous blood gases. Regarding venous hyperoxia, there is a need to differentiate capillary to tissue transport defect (low oxygen utilisation-LOU) from AVS, which may exist simultaneously. The AVS may be associated with normal or relatively high oxygen utilization from the capillary vessels and increased CO2 production. AVS is proposed to carry protective and ‘stealing’ properties including renal, cardiac, and pulmonary hemodynamic. Calculations of the AVS may be important for dynamic assessment of vascular and metabolic status and in emergency medicine. |
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Calculations of the AVS may be important for dynamic assessment of vascular and metabolic status and in emergency medicine.</description><identifier>ISSN: 0960-3271</identifier><identifier>EISSN: 1477-0903</identifier><identifier>DOI: 10.1177/0960327109357216</identifier><identifier>PMID: 20026514</identifier><language>eng</language><publisher>London, England: SAGE Publications</publisher><subject>Algorithms ; Arteriovenous Anastomosis - physiology ; Arteriovenous Malformations - physiopathology ; Biochemistry ; Biological and medical sciences ; Blood and lymphatic vessels ; Capillaries - physiology ; Carbon Dioxide - metabolism ; Cardiology ; Cardiology. Vascular system ; Coronary Vessels - physiopathology ; Defects ; Exercise - physiology ; Hemodynamics ; Humans ; Hyperoxia - etiology ; Kidney - blood supply ; Kidney - physiopathology ; Lung - blood supply ; Lung - physiology ; Medical sciences ; Miscellaneous ; Oxygen Consumption ; Side effects ; Toxicology</subject><ispartof>Human & experimental toxicology, 2010-02, Vol.29 (2), p.141-144</ispartof><rights>The Author(s) 2010</rights><rights>2015 INIST-CNRS</rights><rights>SAGE Publications © Feb 2010</rights><lds50>peer_reviewed</lds50><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c435t-7ad5d7a33d6266747917468f7440f9152cce0b68c8ebdb9525a4feb43ae713813</citedby><cites>FETCH-LOGICAL-c435t-7ad5d7a33d6266747917468f7440f9152cce0b68c8ebdb9525a4feb43ae713813</cites></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><linktopdf>$$Uhttps://journals.sagepub.com/doi/pdf/10.1177/0960327109357216$$EPDF$$P50$$Gsage$$H</linktopdf><linktohtml>$$Uhttps://journals.sagepub.com/doi/10.1177/0960327109357216$$EHTML$$P50$$Gsage$$H</linktohtml><link.rule.ids>314,780,784,21966,27853,27924,27925,44945,45333</link.rule.ids><linktorsrc>$$Uhttps://journals.sagepub.com/doi/full/10.1177/0960327109357216?utm_source=summon&utm_medium=discovery-provider$$EView_record_in_SAGE_Publications$$FView_record_in_$$GSAGE_Publications</linktorsrc><backlink>$$Uhttp://pascal-francis.inist.fr/vibad/index.php?action=getRecordDetail&idt=22463965$$DView record in Pascal Francis$$Hfree_for_read</backlink><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/20026514$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Rozin, Alexander P</creatorcontrib><title>Arterio-venous shunts or low oxygen utilization?</title><title>Human & experimental toxicology</title><addtitle>Hum Exp Toxicol</addtitle><description>An idea of arteriovenous shunts (AVS) was proposed for explanation of dynamic regulation of oxygenation and venous hyperoxia. A formula enabling calculation of AVS and real CO 2 production has recently been derived by comparing data of arterial and venous blood gases. Regarding venous hyperoxia, there is a need to differentiate capillary to tissue transport defect (low oxygen utilisation-LOU) from AVS, which may exist simultaneously. The AVS may be associated with normal or relatively high oxygen utilization from the capillary vessels and increased CO2 production. AVS is proposed to carry protective and ‘stealing’ properties including renal, cardiac, and pulmonary hemodynamic. Calculations of the AVS may be important for dynamic assessment of vascular and metabolic status and in emergency medicine.</description><subject>Algorithms</subject><subject>Arteriovenous Anastomosis - physiology</subject><subject>Arteriovenous Malformations - physiopathology</subject><subject>Biochemistry</subject><subject>Biological and medical sciences</subject><subject>Blood and lymphatic vessels</subject><subject>Capillaries - physiology</subject><subject>Carbon Dioxide - metabolism</subject><subject>Cardiology</subject><subject>Cardiology. Vascular system</subject><subject>Coronary Vessels - physiopathology</subject><subject>Defects</subject><subject>Exercise - physiology</subject><subject>Hemodynamics</subject><subject>Humans</subject><subject>Hyperoxia - etiology</subject><subject>Kidney - blood supply</subject><subject>Kidney - physiopathology</subject><subject>Lung - blood supply</subject><subject>Lung - physiology</subject><subject>Medical sciences</subject><subject>Miscellaneous</subject><subject>Oxygen Consumption</subject><subject>Side effects</subject><subject>Toxicology</subject><issn>0960-3271</issn><issn>1477-0903</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2010</creationdate><recordtype>article</recordtype><recordid>eNp10M1LwzAYBvAgipvTuycpgniqvvlOTjKGXyB40XNJ03R2dM1MWnX-9XZsOhh4yuH9vW8eHoROMVxhLOU1aAGUSAyackmw2ENDzKRMQQPdR8PVOF3NB-goxhkACM3xIRoQACI4ZkME49C6UPn0wzW-i0l865o2Jj4ktf9M_Ndy6pqka6u6-jZt5ZubY3RQmjq6k807Qq93ty-Th_Tp-f5xMn5KLaO8TaUpeCENpYUgQkgmNZZMqFIyBqXGnFjrIBfKKpcXueaEG1a6nFHjJKYK0xG6XN9dBP_eudhm8ypaV9emcX3QTFKqONVc9fJ8R858F5o-XEYIKMGUEj2CNbLBxxhcmS1CNTdhmWHIVl1mu132K2ebu10-d8Xfwm95PbjYABOtqctgGlvFrSNMUC1479K1i2bqtuH-_fgHw6CGMQ</recordid><startdate>201002</startdate><enddate>201002</enddate><creator>Rozin, Alexander P</creator><general>SAGE Publications</general><general>Sage Publications</general><general>Sage Publications Ltd</general><scope>IQODW</scope><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>3V.</scope><scope>7RV</scope><scope>7ST</scope><scope>7TK</scope><scope>7U7</scope><scope>7X7</scope><scope>7XB</scope><scope>88E</scope><scope>8FI</scope><scope>8FJ</scope><scope>8FK</scope><scope>ABUWG</scope><scope>AFKRA</scope><scope>BENPR</scope><scope>C1K</scope><scope>CCPQU</scope><scope>FYUFA</scope><scope>GHDGH</scope><scope>K9.</scope><scope>KB0</scope><scope>M0S</scope><scope>M1P</scope><scope>NAPCQ</scope><scope>PQEST</scope><scope>PQQKQ</scope><scope>PQUKI</scope><scope>SOI</scope><scope>7X8</scope></search><sort><creationdate>201002</creationdate><title>Arterio-venous shunts or low oxygen utilization?</title><author>Rozin, Alexander P</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c435t-7ad5d7a33d6266747917468f7440f9152cce0b68c8ebdb9525a4feb43ae713813</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2010</creationdate><topic>Algorithms</topic><topic>Arteriovenous Anastomosis - physiology</topic><topic>Arteriovenous Malformations - physiopathology</topic><topic>Biochemistry</topic><topic>Biological and medical sciences</topic><topic>Blood and lymphatic vessels</topic><topic>Capillaries - physiology</topic><topic>Carbon Dioxide - metabolism</topic><topic>Cardiology</topic><topic>Cardiology. Vascular system</topic><topic>Coronary Vessels - physiopathology</topic><topic>Defects</topic><topic>Exercise - physiology</topic><topic>Hemodynamics</topic><topic>Humans</topic><topic>Hyperoxia - etiology</topic><topic>Kidney - blood supply</topic><topic>Kidney - physiopathology</topic><topic>Lung - blood supply</topic><topic>Lung - physiology</topic><topic>Medical sciences</topic><topic>Miscellaneous</topic><topic>Oxygen Consumption</topic><topic>Side effects</topic><topic>Toxicology</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Rozin, Alexander P</creatorcontrib><collection>Pascal-Francis</collection><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>ProQuest Central (Corporate)</collection><collection>ProQuest Nursing and Allied Health Journals</collection><collection>Environment Abstracts</collection><collection>Neurosciences Abstracts</collection><collection>Toxicology Abstracts</collection><collection>Health & Medical Collection</collection><collection>ProQuest Central (purchase pre-March 2016)</collection><collection>Medical Database (Alumni Edition)</collection><collection>Hospital Premium Collection</collection><collection>Hospital Premium Collection (Alumni Edition)</collection><collection>ProQuest Central (Alumni) (purchase pre-March 2016)</collection><collection>ProQuest Central (Alumni)</collection><collection>ProQuest Central</collection><collection>AUTh Library subscriptions: ProQuest Central</collection><collection>Environmental Sciences and Pollution Management</collection><collection>ProQuest One Community College</collection><collection>Health Research Premium Collection</collection><collection>Health Research Premium Collection (Alumni)</collection><collection>ProQuest Health & Medical Complete (Alumni)</collection><collection>Nursing & Allied Health Database (Alumni Edition)</collection><collection>Health & Medical Collection (Alumni Edition)</collection><collection>Medical Database</collection><collection>Nursing & Allied Health Premium</collection><collection>ProQuest One Academic Eastern Edition (DO NOT USE)</collection><collection>ProQuest One Academic</collection><collection>ProQuest One Academic UKI Edition</collection><collection>Environment Abstracts</collection><collection>MEDLINE - Academic</collection><jtitle>Human & experimental toxicology</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext_linktorsrc</fulltext></delivery><addata><au>Rozin, Alexander P</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Arterio-venous shunts or low oxygen utilization?</atitle><jtitle>Human & experimental toxicology</jtitle><addtitle>Hum Exp Toxicol</addtitle><date>2010-02</date><risdate>2010</risdate><volume>29</volume><issue>2</issue><spage>141</spage><epage>144</epage><pages>141-144</pages><issn>0960-3271</issn><eissn>1477-0903</eissn><abstract>An idea of arteriovenous shunts (AVS) was proposed for explanation of dynamic regulation of oxygenation and venous hyperoxia. A formula enabling calculation of AVS and real CO 2 production has recently been derived by comparing data of arterial and venous blood gases. Regarding venous hyperoxia, there is a need to differentiate capillary to tissue transport defect (low oxygen utilisation-LOU) from AVS, which may exist simultaneously. The AVS may be associated with normal or relatively high oxygen utilization from the capillary vessels and increased CO2 production. AVS is proposed to carry protective and ‘stealing’ properties including renal, cardiac, and pulmonary hemodynamic. Calculations of the AVS may be important for dynamic assessment of vascular and metabolic status and in emergency medicine.</abstract><cop>London, England</cop><pub>SAGE Publications</pub><pmid>20026514</pmid><doi>10.1177/0960327109357216</doi><tpages>4</tpages><oa>free_for_read</oa></addata></record> |
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subjects | Algorithms Arteriovenous Anastomosis - physiology Arteriovenous Malformations - physiopathology Biochemistry Biological and medical sciences Blood and lymphatic vessels Capillaries - physiology Carbon Dioxide - metabolism Cardiology Cardiology. Vascular system Coronary Vessels - physiopathology Defects Exercise - physiology Hemodynamics Humans Hyperoxia - etiology Kidney - blood supply Kidney - physiopathology Lung - blood supply Lung - physiology Medical sciences Miscellaneous Oxygen Consumption Side effects Toxicology |
title | Arterio-venous shunts or low oxygen utilization? |
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