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Arterio-venous shunts or low oxygen utilization?

An idea of arteriovenous shunts (AVS) was proposed for explanation of dynamic regulation of oxygenation and venous hyperoxia. A formula enabling calculation of AVS and real CO 2 production has recently been derived by comparing data of arterial and venous blood gases. Regarding venous hyperoxia, the...

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Published in:Human & experimental toxicology 2010-02, Vol.29 (2), p.141-144
Main Author: Rozin, Alexander P
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Language:English
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description An idea of arteriovenous shunts (AVS) was proposed for explanation of dynamic regulation of oxygenation and venous hyperoxia. A formula enabling calculation of AVS and real CO 2 production has recently been derived by comparing data of arterial and venous blood gases. Regarding venous hyperoxia, there is a need to differentiate capillary to tissue transport defect (low oxygen utilisation-LOU) from AVS, which may exist simultaneously. The AVS may be associated with normal or relatively high oxygen utilization from the capillary vessels and increased CO2 production. AVS is proposed to carry protective and ‘stealing’ properties including renal, cardiac, and pulmonary hemodynamic. Calculations of the AVS may be important for dynamic assessment of vascular and metabolic status and in emergency medicine.
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subjects Algorithms
Arteriovenous Anastomosis - physiology
Arteriovenous Malformations - physiopathology
Biochemistry
Biological and medical sciences
Blood and lymphatic vessels
Capillaries - physiology
Carbon Dioxide - metabolism
Cardiology
Cardiology. Vascular system
Coronary Vessels - physiopathology
Defects
Exercise - physiology
Hemodynamics
Humans
Hyperoxia - etiology
Kidney - blood supply
Kidney - physiopathology
Lung - blood supply
Lung - physiology
Medical sciences
Miscellaneous
Oxygen Consumption
Side effects
Toxicology
title Arterio-venous shunts or low oxygen utilization?
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