Assessment of enamel hypoplasia in autoimmune polyendocrinopathy-candidiasis-ectodermal dystrophy (APECED)

The features of enamel hypoplasia in a small group of patients with autoimmune polyendocrinopathy‐candidiasis‐ectodermal dystrophy (APECED) are described. Using a recently developed method, the authors evaluated quantitatively the amount of defect in each tooth by measuring the width of the hypoplas...

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Bibliographic Details
Published in:Journal of oral pathology & medicine 1998-07, Vol.27 (6), p.278-282
Main Authors: Perniola, Roberto, Tamborrino, Giuseppe, Marsigliante, Santo, De Rinaldis, Corrado
Format: Article
Language:English
Subjects:
Adult
Age Factors
Bicuspid - pathology
Biological and medical sciences
Child
Cuspid - pathology
Dental Enamel - pathology
Dental Enamel Hypoplasia - pathology
Dentistry
enamel hypoplasia
Endocrinopathies
Female
General aspects. Associated endocrine diseases. Endocrine paraneoplasic syndromes
Humans
hypoparathyroidism
Hypoparathyroidism - pathology
Incisor - pathology
Key Words: APECED
Male
Mandible
Maxilla
Medical sciences
Molar - pathology
Odontometry
Polyendocrinopathies, Autoimmune - pathology
Tooth Cervix - pathology
Tooth Crown - pathology
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Summary:The features of enamel hypoplasia in a small group of patients with autoimmune polyendocrinopathy‐candidiasis‐ectodermal dystrophy (APECED) are described. Using a recently developed method, the authors evaluated quantitatively the amount of defect in each tooth by measuring the width of the hypoplastic lesions and dividing the value by the crown height. They then assessed the degree of damage in each tooth type (from central incisors to second premolars) and patient. Canines were the most severely affected among maxillary and mandibular teeth, but all tooth types were involved. Analysing both the differences between patients and their age at the beginning of the defect, the authors observe that hypoparathyroidism is not responsible for the onset of enamel hypoplasia in APECED, although it may contribute to the damage.
ISSN:0904-2512
1600-0714
DOI:10.1111/j.1600-0714.1998.tb01956.x