Sudden death: Do cytokines and prothrombotic peptides contribute to the occurrence of ventricular fibrillation during acute myocardial infarction?

Abstract Aims Sudden cardiac death (SCD) is frequently caused by ventricular fibrillation (VF) occurring in the course of acute myocardial infarction (AMI). It has not been investigated yet, to what extent markers of coagulation activation and inflammation differ between patients with and without VF...

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Bibliographic Details
Published in:International journal of cardiology 2010-11, Vol.145 (1), p.118-119
Main Authors: Elmas, Elif, Popp, Tatjana, Lang, Siegfried, Dempfle, Carl Erik, Kälsch, Thorsten, Borggrefe, Martin
Format: Article
Language:English
Subjects:
Anesthesia. Intensive care medicine. Transfusions. Cell therapy and gene therapy
Biological and medical sciences
Biomarkers - blood
Blood Coagulation Factors - adverse effects
Blood Coagulation Factors - metabolism
Cardiac dysrhythmias
Cardiology. Vascular system
Cardiovascular
Coagulation
Coronary heart disease
Coronary Thrombosis - blood
Coronary Thrombosis - etiology
Cross-Sectional Studies
Cytokines - blood
Cytokines - physiology
Death, Sudden, Cardiac - etiology
Emergency and intensive care: neonates and children. Prematurity. Sudden death
Female
Heart
Humans
Inflammation
Intensive care medicine
Male
Medical sciences
Middle Aged
Myocardial infarction
Myocardial Infarction - blood
Myocardial Infarction - complications
Myocardial Infarction - mortality
Myocarditis. Cardiomyopathies
Peptides - blood
Peptides - physiology
Prospective Studies
Protein Precursors - adverse effects
Protein Precursors - biosynthesis
Protein Precursors - blood
Retrospective Studies
Sudden cardiac death
Thrombin - adverse effects
Thrombin - biosynthesis
Ventricular Fibrillation - blood
Ventricular Fibrillation - etiology
Ventricular Fibrillation - mortality
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Summary:Abstract Aims Sudden cardiac death (SCD) is frequently caused by ventricular fibrillation (VF) occurring in the course of acute myocardial infarction (AMI). It has not been investigated yet, to what extent markers of coagulation activation and inflammation differ between patients with and without VF in the acute phase of AMI.
ISSN:0167-5273
1874-1754
DOI:10.1016/j.ijcard.2009.06.014