Cardiovascular consequences of severe hypophosphataemia in brain-dead patients

Hypophosphataemia is known to induce reversible myocardial dysfunction, but the incidence of hypophosphataemia and its effect on myocardial function during brain death are unknown. In 90 consecutive brain-dead patients, we measured plasma concentrations of phosphate and left ventricular ejection fra...

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Bibliographic Details
Published in:British Journal of Anaesthesia 1995-04, Vol.74 (4), p.424-429
Main Authors: RIOU, B., KALFON, P., AROCK, M., GOARIN, J.-P., SAADA, M., VIARS, P.
Format: Article
Language:English
Subjects:
Adolescent
Adult
Anesthesia. Intensive care medicine. Transfusions. Cell therapy and gene therapy
Biological and medical sciences
Brain Death - blood
Brain Death - physiopathology
Brain Death - urine
Child
Clinical death. Palliative care. Organ gift and preservation
Complications
death
Female
Heart
Humans
hypophosphataemia
Hypophosphatemia - complications
Hypophosphatemia - physiopathology
Ions
Male
Medical sciences
Middle Aged
myocardial function
Oxygen - blood
Oxygen Consumption
phosphate
Phosphates - urine
Phosphorus - administration & dosage
Prospective Studies
Stroke Volume
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Summary:Hypophosphataemia is known to induce reversible myocardial dysfunction, but the incidence of hypophosphataemia and its effect on myocardial function during brain death are unknown. In 90 consecutive brain-dead patients, we measured plasma concentrations of phosphate and left ventricular ejection fraction area (LVEFa), using transoesophageal echocardiography. In 15 severely hypophosphataemic (
ISSN:1471-6771
0007-0912
0007-0912
1471-6771
DOI:10.1093/bja/74.4.424