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Status epilepticus presenting as progressive dysphasia
Status epilepticus is usually a straightforward diagnosis when a patient has two or more seizures without regaining consciousness. However, when status is non-convulsive and, in particular, has a temporal lobe flavour the clinical presentation may be misleading. Presentation with automatic or psychi...
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Published in: | Neuroradiology 1995-08, Vol.37 (6), p.438-439 |
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container_issue | 6 |
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container_title | Neuroradiology |
container_volume | 37 |
creator | MURCHISON, J. T SELLAR, R. J STEERS, A. J. W |
description | Status epilepticus is usually a straightforward diagnosis when a patient has two or more seizures without regaining consciousness. However, when status is non-convulsive and, in particular, has a temporal lobe flavour the clinical presentation may be misleading. Presentation with automatic or psychic behaviour is well recorded. We report a patient with nonconvulsive status who presented with progressive dysphasia with widespread CT and MRI changes. The dysphasia and imaging changes led to a diagnosis of a probable neoplastic brain process but reversed with anticonvulsant treatment. |
doi_str_mv | 10.1007/BF00600083 |
format | article |
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T</creatorcontrib><creatorcontrib>SELLAR, R. J</creatorcontrib><creatorcontrib>STEERS, A. J. W</creatorcontrib><title>Status epilepticus presenting as progressive dysphasia</title><title>Neuroradiology</title><addtitle>Neuroradiology</addtitle><description>Status epilepticus is usually a straightforward diagnosis when a patient has two or more seizures without regaining consciousness. However, when status is non-convulsive and, in particular, has a temporal lobe flavour the clinical presentation may be misleading. Presentation with automatic or psychic behaviour is well recorded. We report a patient with nonconvulsive status who presented with progressive dysphasia with widespread CT and MRI changes. The dysphasia and imaging changes led to a diagnosis of a probable neoplastic brain process but reversed with anticonvulsant treatment.</description><subject>Adult</subject><subject>Anticonvulsants - administration & dosage</subject><subject>Aphasia - diagnosis</subject><subject>Aphasia - etiology</subject><subject>Aphasia - physiopathology</subject><subject>Biological and medical sciences</subject><subject>Brain Diseases - complications</subject><subject>Brain Diseases - diagnosis</subject><subject>Brain Diseases - physiopathology</subject><subject>Cysts - complications</subject><subject>Cysts - diagnosis</subject><subject>Cysts - physiopathology</subject><subject>Diagnosis, Differential</subject><subject>Electroencephalography - drug effects</subject><subject>Female</subject><subject>Headache. Facial pains. Syncopes. Epilepsia. Intracranial hypertension. Brain oedema. Cerebral palsy</subject><subject>Humans</subject><subject>Magnetic Resonance Imaging</subject><subject>Medical sciences</subject><subject>Nervous system (semeiology, syndromes)</subject><subject>Neurology</subject><subject>Status Epilepticus - complications</subject><subject>Status Epilepticus - diagnosis</subject><subject>Status Epilepticus - physiopathology</subject><subject>Temporal Lobe - pathology</subject><subject>Temporal Lobe - physiopathology</subject><subject>Tomography, X-Ray Computed</subject><issn>0028-3940</issn><issn>1432-1920</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>1995</creationdate><recordtype>article</recordtype><recordid>eNpFkEFLw0AQhRdRaq1evAs5iAchOruT7G6OWqwKBQ_qOWw227qSJjGTCP33bmmozGFm3nw8hsfYJYc7DqDuHxcAEgA0HrEpT1DEPBNwzKYAQseYJXDKzoi-A4IK1YRNVKKUTrIpk--96QeKXOsr1_behrntHLm69_U6MrutWQeB_K-Lyi21X4a8OWcnK1ORuxj7jH0unj7mL_Hy7fl1_rCMLXLexzJJtSm1FFAI4DZFoUtRSmesSQVYU_ACsyClTu8KC6mDiBZliZihwBm72fuGL34GR32-8WRdVZnaNQPlSqUZYCoDeLsHbdcQdW6Vt53fmG6bc8h3IeX_IQX4anQdio0rD-iYSrhfj3dD1lSrztTW0wFDicCFwj_jQm2I</recordid><startdate>19950801</startdate><enddate>19950801</enddate><creator>MURCHISON, J. 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W</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c311t-6458ad8620b201c5328d2d6eaca520cab1b398d25e8e8e83b68cab3c36d339323</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>1995</creationdate><topic>Adult</topic><topic>Anticonvulsants - administration & dosage</topic><topic>Aphasia - diagnosis</topic><topic>Aphasia - etiology</topic><topic>Aphasia - physiopathology</topic><topic>Biological and medical sciences</topic><topic>Brain Diseases - complications</topic><topic>Brain Diseases - diagnosis</topic><topic>Brain Diseases - physiopathology</topic><topic>Cysts - complications</topic><topic>Cysts - diagnosis</topic><topic>Cysts - physiopathology</topic><topic>Diagnosis, Differential</topic><topic>Electroencephalography - drug effects</topic><topic>Female</topic><topic>Headache. Facial pains. Syncopes. Epilepsia. Intracranial hypertension. Brain oedema. Cerebral palsy</topic><topic>Humans</topic><topic>Magnetic Resonance Imaging</topic><topic>Medical sciences</topic><topic>Nervous system (semeiology, syndromes)</topic><topic>Neurology</topic><topic>Status Epilepticus - complications</topic><topic>Status Epilepticus - diagnosis</topic><topic>Status Epilepticus - physiopathology</topic><topic>Temporal Lobe - pathology</topic><topic>Temporal Lobe - physiopathology</topic><topic>Tomography, X-Ray Computed</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>MURCHISON, J. T</creatorcontrib><creatorcontrib>SELLAR, R. J</creatorcontrib><creatorcontrib>STEERS, A. J. 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W</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Status epilepticus presenting as progressive dysphasia</atitle><jtitle>Neuroradiology</jtitle><addtitle>Neuroradiology</addtitle><date>1995-08-01</date><risdate>1995</risdate><volume>37</volume><issue>6</issue><spage>438</spage><epage>439</epage><pages>438-439</pages><issn>0028-3940</issn><eissn>1432-1920</eissn><coden>NRDYAB</coden><abstract>Status epilepticus is usually a straightforward diagnosis when a patient has two or more seizures without regaining consciousness. However, when status is non-convulsive and, in particular, has a temporal lobe flavour the clinical presentation may be misleading. Presentation with automatic or psychic behaviour is well recorded. We report a patient with nonconvulsive status who presented with progressive dysphasia with widespread CT and MRI changes. 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language | eng |
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source | Springer LINK Archives |
subjects | Adult Anticonvulsants - administration & dosage Aphasia - diagnosis Aphasia - etiology Aphasia - physiopathology Biological and medical sciences Brain Diseases - complications Brain Diseases - diagnosis Brain Diseases - physiopathology Cysts - complications Cysts - diagnosis Cysts - physiopathology Diagnosis, Differential Electroencephalography - drug effects Female Headache. Facial pains. Syncopes. Epilepsia. Intracranial hypertension. Brain oedema. Cerebral palsy Humans Magnetic Resonance Imaging Medical sciences Nervous system (semeiology, syndromes) Neurology Status Epilepticus - complications Status Epilepticus - diagnosis Status Epilepticus - physiopathology Temporal Lobe - pathology Temporal Lobe - physiopathology Tomography, X-Ray Computed |
title | Status epilepticus presenting as progressive dysphasia |
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