Viral arthritides

Viruses are common etiologic agents of acute polyarthritis worldwide. Rheumatic symptoms are often accompanied by other viral symptoms such as fever, rash, myalgia or nausea, but isolated arthritis may occur resembling or imitating early rheumatoid arthritis. In Europe and North America, the most co...

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Bibliographic Details
Published in:Zeitschrift für Rheumatologie 2010-12, Vol.69 (10), p.871-878
Main Authors: Märker-Hermann, E, Schütz, N, Bauer, H
Format: Article
Language:ger
Subjects:
Alphavirus - immunology
Alphavirus Infections - complications
Alphavirus Infections - diagnosis
Alphavirus Infections - immunology
Arthritis - diagnosis
Arthritis - etiology
Arthritis - immunology
Arthritis, Rheumatoid - diagnosis
Arthritis, Rheumatoid - etiology
Arthritis, Rheumatoid - immunology
Diagnosis, Differential
Hepatitis B - complications
Hepatitis B - diagnosis
Hepatitis B - immunology
Hepatitis C - complications
Hepatitis C - diagnosis
Hepatitis C - immunology
HTLV-I Infections - complications
HTLV-I Infections - diagnosis
HTLV-I Infections - immunology
Humans
Parvoviridae Infections - complications
Parvoviridae Infections - diagnosis
Parvoviridae Infections - immunology
Parvovirus B19, Human - immunology
Rubella - complications
Rubella - diagnosis
Rubella - immunology
Virus Diseases - complications
Virus Diseases - diagnosis
Virus Diseases - immunology
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Description
Summary:Viruses are common etiologic agents of acute polyarthritis worldwide. Rheumatic symptoms are often accompanied by other viral symptoms such as fever, rash, myalgia or nausea, but isolated arthritis may occur resembling or imitating early rheumatoid arthritis. In Europe and North America, the most common arthritogenic viruses include parvovirus B19, rubella, hepatitis B and C, whereas mosquito-borne alpha-viruses are endemic in Africa, Australia, the western Pacific and South America. The latter have attracted the interest of European rheumatologists due to the increasing number of tourists travelling to these regions. Viruses may cause arthritis via various mechanisms, including direct synovial infection leading to cell lysis, immune complex formation or pro-inflammatory cytokine induction. Indirect viral pathogenic effects may induce autoimmune reactions via bystander activation or epitope spreading.
ISSN:1435-1250
DOI:10.1007/s00393-010-0701-6