Epinephrine induced hyperglycemia in bulls and its relationship to polioencephalomalacia

Data on blood glucose concentration in bulls affected with molasses associated polioencephalomalacia are controversial. It has been suggested that the brain lesions are related to a "hypoglycemic state" during the development of polioencephalomalacia. This paper reports the mobilization of...

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Bibliographic Details
Published in:Canadian journal of comparative medicine 1975-07, Vol.39 (3), p.321-323
Main Authors: Mella, C.M, Margolles, E, Loew, F.M
Format: Article
Language:English
Subjects:
animal diseases
animal health
Animals
Blood Glucose - analysis
Cattle
Cattle Diseases - etiology
Cattle Diseases - physiopathology
Encephalomalacia - etiology
Encephalomalacia - physiopathology
Encephalomalacia - veterinary
Epinephrine
Hyperglycemia - veterinary
Hypoglycemia - complications
Hypoglycemia - veterinary
Male
noninfectious diseases
Plant Poisoning - veterinary
Regression Analysis
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Summary:Data on blood glucose concentration in bulls affected with molasses associated polioencephalomalacia are controversial. It has been suggested that the brain lesions are related to a "hypoglycemic state" during the development of polioencephalomalacia. This paper reports the mobilization of glucose by means of the epinephrine test in three bulls fed two diets, one forage based and the other molasses based. The results showed significantly greater hyperglycemic responses in the animals during the molasses diet than during the forage one. This probably means that glucose stores (as glycogen) are higher in cattle consuming molasses than those consuming forage. Such hepatic glucose output is in disagreement with the hypoglycemia theory as the cause of the early stages of brain lesions and focuses the probable cause as being related to glucose utilization.
ISSN:0008-4050