Serum cardiac troponin T and plasma brain natriuretic peptide in patients with cardiac decompensation

In our earlier study, idiopathic dilated cardiomyopathy patients with persistently high serum troponin T (TnT) concentrations had notably depressed left ventricular ejection fractions, increased left ventricular diastolic dimensions, and adverse long term outcomes. 1- 3 Therefore, TnT seems to be a...

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Bibliographic Details
Published in:British heart journal 2002-12, Vol.88 (6), p.647-648
Main Authors: Sato, Y, Taniguchi, R, Makiyama, T, Nagai, K, Okada, H, Yamada, T, Matsumori, A, Takatsu, Y
Format: Article
Language:English
Subjects:
Aged
Angina pectoris
Biological and medical sciences
Biomarkers - blood
BNP
brain natriuretic peptide
cardiac decompensation
Cardiology. Vascular system
Cardiomyopathy
CHF
congestive heart failure
Diagnosis
Female
Heart
Heart attacks
Heart diseases
Heart failure
Heart Failure - blood
Heart Failure - etiology
Heart failure, cardiogenic pulmonary edema, cardiac enlargement
Humans
Male
Medical sciences
Natriuretic Peptide, Brain - blood
Patients
Peptides
Physiological aspects
Plasma
Scientific Letters
TnT
troponin T
Troponin T - blood
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Summary:In our earlier study, idiopathic dilated cardiomyopathy patients with persistently high serum troponin T (TnT) concentrations had notably depressed left ventricular ejection fractions, increased left ventricular diastolic dimensions, and adverse long term outcomes. 1- 3 Therefore, TnT seems to be a marker of ongoing subclinical myocyte injury, although the mechanism of its release is unknown. No patient had a recent myocardial infarction or unstable angina pectoris within one year, and no electrocardiographic changes or increases in creatine kinase enzyme were observed throughout the study period.
ISSN:1355-6037
0007-0769
1468-201X
DOI:10.1136/heart.88.6.647