Uterine arteriovenous malformation

Uterine arteriovenous malformation (AVM) is a little known condition of which, to date, very few cases have been described. It has a very diverse symptomatology, even though in most cases, it is diagnosed during a severe and acute haemorrhagic event. Its treatment can vary from expectant management...

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Bibliographic Details
Published in:BMJ case reports 2013-02, Vol.2013, p.bcr2012008443
Main Authors: Sellers, Francisco, Palacios-Marqués, Ana, Moliner, Belen, Bernabeu, Rafael
Format: Article
Language:English
Subjects:
Abortion
Abortion, Spontaneous - blood
Abortion, Spontaneous - diagnostic imaging
Adult
Angiogenesis
Arteriovenous Malformations - blood
Arteriovenous Malformations - diagnostic imaging
Asymptomatic
Asymptomatic Diseases
Chorionic Gonadotropin, beta Subunit, Human - blood
Endometrium
Europe (West)
Female
Flow velocity
Growth factors
Gynecology
Humans
Miscarriage
Pathogenesis
Pregnancy
Rare Disease
Ultrasonic imaging
Ultrasonography, Doppler, Color
Uterine Artery - abnormalities
Uterine Artery - diagnostic imaging
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Summary:Uterine arteriovenous malformation (AVM) is a little known condition of which, to date, very few cases have been described. It has a very diverse symptomatology, even though in most cases, it is diagnosed during a severe and acute haemorrhagic event. Its treatment can vary from expectant management to hysterectomy; however, current evidence suggests that the embolisation of uterine arteries is the most effective approach, especially if fertility is to be preserved. We present a case report classified as AVM, with additional images that show the appearance of this pathology in a short span of time. This case has a number of peculiarities: unusual persistence of human chorionic gonadotropin hormone (β-HCG), asymptomatic patient, quick establishment of the lesion and its duration with unchanging characteristics and finally its spontaneous resolution without further consequences. This entity shows an aetiopathogenesis, that is, not well established or described. We discuss its physiopathology and aetiopathogenesis.
ISSN:1757-790X
1757-790X
DOI:10.1136/bcr-2012-008443