Non-prion-type transmission in A53T α-synuclein transgenic mice: a normal component of spinal homogenates from naïve non-transgenic mice induces robust α-synuclein pathology

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Published in:Acta neuropathologica 2016, Vol.131 (1), p.151-154
Main Authors: Sacino, Amanda N., Ayers, Jacob I., Brooks, Mieu M. T., Chakrabarty, Paramita, Hudson, Vincent J., Howard, Jasie K., Golde, Todd E., Giasson, Benoit I., Borchelt, David R.
Format: Article
Language:English
Subjects:
alpha-Synuclein - genetics
alpha-Synuclein - metabolism
Animals
Correspondence
Humans
Medicine
Medicine & Public Health
Mice, Transgenic
Neurosciences
Pathology
Prions - metabolism
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cited_by cdi_FETCH-LOGICAL-c4271-c29cdd8255a3d472ad35f506704bc35e83270c8eaabc4bdebfd25c555f1053e3
cites cdi_FETCH-LOGICAL-c4271-c29cdd8255a3d472ad35f506704bc35e83270c8eaabc4bdebfd25c555f1053e3
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container_title Acta neuropathologica
container_volume 131
creator Sacino, Amanda N.
Ayers, Jacob I.
Brooks, Mieu M. T.
Chakrabarty, Paramita
Hudson, Vincent J.
Howard, Jasie K.
Golde, Todd E.
Giasson, Benoit I.
Borchelt, David R.
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doi_str_mv 10.1007/s00401-015-1505-1
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subjects alpha-Synuclein - genetics
alpha-Synuclein - metabolism
Animals
Correspondence
Humans
Medicine
Medicine & Public Health
Mice, Transgenic
Neurosciences
Pathology
Prions - metabolism
title Non-prion-type transmission in A53T α-synuclein transgenic mice: a normal component of spinal homogenates from naïve non-transgenic mice induces robust α-synuclein pathology
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