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Lack of Gdf11 does not improve anemia or prevent the activity of RAP-536 in a mouse model of β-thalassemia
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Published in: | Blood 2019-08, Vol.134 (6), p.568-572 |
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creator | Guerra, Amaliris Oikonomidou, Paraskevi Rea Sinha, Sayantani Zhang, Jianbing Lo Presti, Vania Hamilton, Callum R. Breda, Laura Casu, Carla La, Ping Martins, Ana C. Sendamarai, Anoop K. Fleming, Mark Rivella, Stefano |
description | There is a Blood Commentary on this article in this issue. |
doi_str_mv | 10.1182/blood.2019001057 |
format | article |
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blood ; Anemia - drug therapy ; Anemia - etiology ; Animals ; beta-Thalassemia - blood ; beta-Thalassemia - drug therapy ; beta-Thalassemia - genetics ; beta-Thalassemia - metabolism ; Biomarkers ; Bone Morphogenetic Proteins - deficiency ; Bone Morphogenetic Proteins - genetics ; Bone Morphogenetic Proteins - metabolism ; Disease Models, Animal ; Growth Differentiation Factors - deficiency ; Growth Differentiation Factors - genetics ; Growth Differentiation Factors - metabolism ; Letter to ; Mice ; Mice, Transgenic ; Recombinant Fusion Proteins - pharmacology ; Treatment Outcome</subject><ispartof>Blood, 2019-08, Vol.134 (6), p.568-572</ispartof><rights>2019 American Society of Hematology</rights><rights>2019 by The American Society of Hematology 2019</rights><lds50>peer_reviewed</lds50><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c377t-60941424a7f4c4aedcf417d7735ebc4cbfec8fb27050f0d8abd9084e3a59f9a43</citedby><cites>FETCH-LOGICAL-c377t-60941424a7f4c4aedcf417d7735ebc4cbfec8fb27050f0d8abd9084e3a59f9a43</cites><orcidid>0000-0002-0938-6558 ; 0000-0002-6602-1854</orcidid></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><linktohtml>$$Uhttps://www.sciencedirect.com/science/article/pii/S000649712042364X$$EHTML$$P50$$Gelsevier$$Hfree_for_read</linktohtml><link.rule.ids>230,314,780,784,885,3547,27922,27923,45778</link.rule.ids><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/31151988$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Guerra, Amaliris</creatorcontrib><creatorcontrib>Oikonomidou, Paraskevi Rea</creatorcontrib><creatorcontrib>Sinha, Sayantani</creatorcontrib><creatorcontrib>Zhang, Jianbing</creatorcontrib><creatorcontrib>Lo Presti, Vania</creatorcontrib><creatorcontrib>Hamilton, Callum R.</creatorcontrib><creatorcontrib>Breda, Laura</creatorcontrib><creatorcontrib>Casu, Carla</creatorcontrib><creatorcontrib>La, Ping</creatorcontrib><creatorcontrib>Martins, Ana C.</creatorcontrib><creatorcontrib>Sendamarai, Anoop K.</creatorcontrib><creatorcontrib>Fleming, Mark</creatorcontrib><creatorcontrib>Rivella, Stefano</creatorcontrib><title>Lack of Gdf11 does not improve anemia or prevent the activity of RAP-536 in a mouse model of β-thalassemia</title><title>Blood</title><addtitle>Blood</addtitle><description>There is a Blood Commentary on this article in this issue.</description><subject>Anemia - 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subjects | Anemia - blood Anemia - drug therapy Anemia - etiology Animals beta-Thalassemia - blood beta-Thalassemia - drug therapy beta-Thalassemia - genetics beta-Thalassemia - metabolism Biomarkers Bone Morphogenetic Proteins - deficiency Bone Morphogenetic Proteins - genetics Bone Morphogenetic Proteins - metabolism Disease Models, Animal Growth Differentiation Factors - deficiency Growth Differentiation Factors - genetics Growth Differentiation Factors - metabolism Letter to Mice Mice, Transgenic Recombinant Fusion Proteins - pharmacology Treatment Outcome |
title | Lack of Gdf11 does not improve anemia or prevent the activity of RAP-536 in a mouse model of β-thalassemia |
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