Cyclooxygenase-2 is critical for the propagation of β-amyloid protein and reducing the glycosylation of tau in Alzheimer’s disease

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Published in:Cellular & molecular immunology 2019-11, Vol.16 (11), p.892-894
Main Authors: Guan, Pei-Pei, Yu, Xin, Zou, Yan-Hui, Wang, Pu
Format: Article
Language:English
Subjects:
631/250/256/2515
692/53/2423
Alzheimer Disease - genetics
Alzheimer Disease - immunology
Alzheimer Disease - pathology
Alzheimer's disease
Amyloid beta-Peptides - genetics
Amyloid beta-Peptides - immunology
Animals
Antibodies
Biomedical and Life Sciences
Biomedicine
Correspondence
Cyclooxygenase 2 - genetics
Cyclooxygenase 2 - immunology
Cyclooxygenase-2
Disease Models, Animal
Glycosylation
Immunology
Medical Microbiology
Mice
Mice, Transgenic
Microbiology
Tau protein
tau Proteins - genetics
tau Proteins - immunology
Vaccine
β-Amyloid
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subjects 631/250/256/2515
692/53/2423
Alzheimer Disease - genetics
Alzheimer Disease - immunology
Alzheimer Disease - pathology
Alzheimer's disease
Amyloid beta-Peptides - genetics
Amyloid beta-Peptides - immunology
Animals
Antibodies
Biomedical and Life Sciences
Biomedicine
Correspondence
Cyclooxygenase 2 - genetics
Cyclooxygenase 2 - immunology
Cyclooxygenase-2
Disease Models, Animal
Glycosylation
Immunology
Medical Microbiology
Mice
Mice, Transgenic
Microbiology
Tau protein
tau Proteins - genetics
tau Proteins - immunology
Vaccine
β-Amyloid
title Cyclooxygenase-2 is critical for the propagation of β-amyloid protein and reducing the glycosylation of tau in Alzheimer’s disease
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