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Impact of time delay on the dynamics of SEIR epidemic model using cellular automata
The delay of an infectious disease is significant when aiming to predict its strength and spreading patterns. In this paper the SEIR (susceptible–exposed–infected–recovered) epidemic spread with time delay is analyzed through a two-dimensional cellular automata model. The time delay corresponding t...
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Published in: | Physica A 2017-04, Vol.471, p.114-125 |
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Main Authors: | , |
Format: | Article |
Language: | English |
Subjects: | |
Citations: | Items that this one cites Items that cite this one |
Online Access: | Get full text |
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Summary: | The delay of an infectious disease is significant when aiming to predict its strength and spreading patterns. In this paper the SEIR (susceptible–exposed–infected–recovered) epidemic spread with time delay is analyzed through a two-dimensional cellular automata model. The time delay corresponding to the infectious span, predominantly, includes death during the latency period in due course of infection. The advancement of whole system is described by SEIR transition function complemented with crucial factors like inhomogeneous population distribution, birth and disease independent mortality. Moreover, to reflect more realistic population dynamics some stochastic parameters like population movement and connections at local level are also considered. The existence and stability of disease free equilibrium is investigated. Two prime behavioral patterns of disease dynamics is found depending on delay. The critical value of delay, beyond which there are notable variations in spread patterns, is computed. The influence of important parameters affecting the disease dynamics on basic reproduction number is also examined. The results obtained show that delay plays an affirmative role to control disease progression in an infected host.
•We presented two dimensional cellular automata model to simulate SEIR epidemic spread with time delay.•The existence and stability of disease free equilibrium is examined.•The behavioral patterns of disease dynamics are explored.•The critical value of delay, beyond which there are notable variations in diffusion patterns, is computed.•The basic reproductive number of the system and its relationship with the vital parameters has been investigated. |
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ISSN: | 0378-4371 1873-2119 |
DOI: | 10.1016/j.physa.2016.12.010 |