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Elevated KIF2C Expression Drives Osteosarcoma Progression by Modulating the Wnt/β‐Catenin Signaling Pathway and Contributing to an Immunosuppressive Tumor Microenvironment

Background Although kinesin family member 2C (KIF2C) is implicated in various cancers, its role in osteosarcoma (OS) and the associated inflammatory microenvironment remains unclear. Methods Publicly available datasets were analyzed to determine KIF2C expression, diagnostic value, and prognostic rel...

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Bibliographic Details
Published in:	Cancer medicine (Malden, MA) MA), 2025-05, Vol.14 (9), p.e70915-n/a
Main Authors:	Liu, Ya‐Yun, Sun, Wu, Liu, Lin, Cheng, Jin‐Hui, Li, Jing‐Tang, Huang, Zu‐Tai, Ouyang, Min
Format:	Article
Language:	English
Subjects:	Animals Antibodies Antigens Apoptosis beta Catenin - metabolism biomarker Bone cancer Bone Neoplasms - genetics Bone Neoplasms - immunology Bone Neoplasms - metabolism Bone Neoplasms - mortality Bone Neoplasms - pathology Bones Cancer therapies Catenin Cell growth Cell Line, Tumor Cell migration Cell Movement Cell Proliferation Chemotherapy Chromosomes clinical significance Datasets Disease Progression Female Gene expression Gene Expression Regulation, Neoplastic Genomes Humans Immunoprecipitation Kinesin kinesin family member 2C (KIF2C) Kinesins - antagonists & inhibitors Kinesins - chemistry Kinesins - genetics Kinesins - metabolism Liver cancer Male Medical prognosis Metastasis Mice Molecular Docking Simulation Molecules Osteosarcoma Osteosarcoma - genetics Osteosarcoma - immunology Osteosarcoma - metabolism Osteosarcoma - mortality Osteosarcoma - pathology Prognosis Signal transduction Tumor microenvironment Tumor Microenvironment - immunology Tumors Wnt protein Wnt Signaling Pathway
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